Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Tuesday, May 7, 2019

Stress-related disorders increase risk for CVD

So because your doctor failed at getting you 100% recovered your stress level shot through the roof possibly leading to the 23% chance of stroke survivors getting PTSD June 2012. What is your doctors solution to prevent this possibility? This is not treatment for PTSD, it is preventing PTSD.  If none, call the hospital president and ask when they will start employing competent doctors.

Stress-related disorders increase risk for CVD

Patients with stress-related disorders including PTSD, adjustment disorder, acute stress reaction and other stress reactions had an increased risk for CVD compared with those without disorders, according to a study published in The BMJ.
“These sudden cardiovascular events carry a high risk of a fatal outcome, so increased clinical awareness of these risks among patients with recently diagnosed stress-related disorders deserves further attention,” Huan Song, MD, PhD, postdoctoral fellow at the Center of Public Health Sciences at University of Iceland in Reykjavik and the department of medical epidemiology and biostatistics at Karolinska Institutet in Stockholm, and colleagues wrote.
Swedish patients with stress disorders
Researchers analyzed data from 136,637 patients (median age, 35 years; 38% men) from the Swedish National Patient Register who did not have CVD before their diagnosis of a stress-related disorder between 1987 and 2013. A cohort of 171,314 siblings (median age, 36 years; 51% men) without stress-related disorders or CVD were also included in the study, in addition to 1,366,370 matched patients (median age, 35 years; 38% men) from the general population who were free from stress-related disorders and CVD.
All patients were followed up from the index date to the first primary diagnosis of CVD, emigration, death or Dec. 31, 2013, whichever occurred first.
Patients with stress-related disorders were followed up for a median of 6.2 years. Follow-up for siblings was conducted for a median of 6.9 years and a median of 6.5 years for the matched cohort.

Patients with stress-related disorders including PTSD, adjustment disorder, acute stress reaction and other stress reactions had an increased risk for CVD compared with those without disorders, according to a study published in The BMJ.
Source: Adobe Stock
The crude incidence of any CVD was 10.5 per 1,000 person-years in patients with a stress-related disorder, 8.4 per 1,000 person-years for their siblings without the disorder and 6.9 per 1,000 person-years in the matched cohort during up to 27 years of follow-up.
When patients with stress-related disorders and their siblings were compared, the HR for any CVD was 1.64 (95% CI, 1.45-1.84). The highest HR was seen for HF during the first year after a diagnosis of a stress-related disorder (HR = 6.95; 95% CI, 1.88-25.68). Overall HRs decreased after 1 year (HR = 1.29; 95% CI, 1.24-1.34) and ranged from 1.12 for arrhythmia (95% CI, 1.04-1.21) to 2.02 for artery thrombosis/embolus (95% CI, 1.45-2.82).
Results were similar in analyses of the matched cohort for any CVD diagnosis during the first year of follow-up (HR = 1.71; 95% CI, 1.59-1.83) and thereafter (HR = 1.36; 95% CI, 1.33-1.3
9).

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