Rehabilitation of Younger Patients Post Stroke

You can read the 53 pages yourself or you can trust your doctor to have absorbed them and created stroke protocols. ROFLMAO.
Rehabilitation of Younger Patients Post Stroke

The effect of change in fasting glucose on the risk of myocardial infarction, stroke, and all-cause mortality: A nationwide cohort study

Your doctor can explain exactly what this means and the protocols you will be following to accomplish. Oops, ROFLMAO, that will never occur. 
https://www.mdlinx.com/internal-medicine/medical-news-article/2018/04/26/prediabetes-blood-glucose-myocardial-infarction-stroke/7509897/?

Cardiovascular Diabetology — | April 18, 2018

Lee G, et al. - Researchers looked at a representative large population and focused on the link between changes in fasting serum glucose and incident cardiovascular disease and all-cause mortality. They found that, in a non-diabetic population, increasing fasting glucose was related to increased risks of myocardial infarction (MI), stroke, and all-cause mortality.

sponsor

Methods

• Data from a retrospective cohort of the Korean National Health Insurance Service was analyzed.
• Study participants included 260,487 Korean adults aged over 40 years without diabetes mellitus and cardiovascular disease at baseline.
• Fasting glucose status was categorized as normal fasting glucose (NFG, fasting glucose: < 100 mg/dL), impaired fasting glucose (IFG, fasting glucose: 100.0–125.9 mg/dL), and diabetic fasting glucose (DFG, fasting glucose: ≥ 126.0 mg/dL)
• Cox proportional hazards regression analyses were performed in the changed group vs the persistently unchanged group (i.e. NFG to NFG or IFG to IFG) in order to obtain the hazards ratio (HR) with 95% confidence interval (CI) for subsequent median 8-year MI, stroke, and all-cause mortality.

• See Also: Stroke type and severity in patients with sub-clinical atrial fibrillation: An analysis from the ASSERT trial

Results

• An increased risk of stroke was noted for individuals who shifted from NFG to DFG (HR [95% CI]: 1.19 [1.02–1.38]).
• Those who shifted from NFG to IFG or DFG saw increased risks of all-cause mortality (HR [95% CI]: 1.08 [1.02–1.14] for NFG to IFG, and 1.56 [1.39–1.75] for NFG to DFG) vs individuals with persistent NFG.
• Participants who shifted from IFG to DFG had an increased risk of MI and all-cause mortality (HR [95% CI]: 1.65 [1.20–2.27] and 1.16 [1.02–1.33], respectively) vs individuals with persistent IFG.

• See Also: Coronary heart disease, heart failure, and the risk of dementia: A systematic review and meta-analysis

Read the full article on Cardiovascular Diabetology

Recommended For You

Recovery hope for paralytic stroke patients from Robot Hand

You can have your doctor analyze this robotic hand vs. the other ones out there. Your doctor should have no problem comparing them since they will be up-to-date on all stroke research. ROFLMAO.

Recovery hope for paralytic stroke patients from Robot Hand

By Arabinda Panda  |  Express News Service  |   Published: 15th April 2018 04:06 AM  |  

Last Updated: 15th April 2018 04:06 AM  |   A+A A-   |  

Shifa treating Shakuntala with the help of robotic hand at SVNIRTAR at Olatpur | Express

CUTTACK: The patients suffering from paralysis after a stroke can now find hope in early recovery. A robotic hand developed by a student of Swami Vivekananda National Institute of Rehabilitation Training and Research (SVNIRTAR) at Olatpur, helps in restoring the hand functions after the stroke.The prototype of the device developed by Shifa Khan, pursuing Masters in Physiotherapy (MPT) of the institution under the guidance of Dr PP Mohanty and Dr Monalisha Patnaik, has been tried on 30 Hemiplegia patients successfully in the last two years. Hemiplegia occurs due to brain stroke usually paralysing one side of the body. The upper limbs are more affected than the lower limbs and the hand function is worst affected, said Shifa.

Robotic hand has been found to be quite effective in restoration of the hand-function in the management of persons with Hemiplegia, she claimed. The Robotic Hand is not only simple to operate but also cost effective, she added. “It is made of an embedded controller and a robotic hand module, which provide patients assistive power to perform hand movements and gradually boost re-learning in the brain of stroke patients,” said Khan.

According to Shifa, once the robotic hand is attached to the patient’s fingers and hand, it begins to detect his or her intention to move through signals from the Hemiplegic side measured by surface electromyography. A nervous response then sets the robotic hand in motion to move the patient’s stiff hand at his or her own will. The device affords substantial flexibility, as each finger assembly can be adjusted to fit different finger lengths.

Unlike the case with conventional rehabilitation treatment, the assistive power from the robotic hand helps the person overcome muscle and joint stiffness, thus allowing him to open or close a paralysed hand or pick up items according to his necessity. Through repetitive exercises, the hand motions send feedback to the patient’s brain, helping it rewire itself in response to new experiences, she said.  The device has been developed by Shifa in collaboration with Eidetic InfoTech Private Limited. A woman patient Shakuntala Palai, undergoing treatment, said the robotic hand has helped her move her fingers  and hopes to use her hand normally soon.

Red meat linked to increased stroke risk

Be careful out there.  2 years old so your doctor has already informed you of this risk I'm sure. ROFLMAO. Not going to change my habits because I can't cut red meat on my own and I dislike asking for help.
https://www.reuters.com/article/us-health-meat-stroke-risk/red-meat-linked-to-increased-stroke-risk-idUSKBN0TE2IA20151125

Lisa Rapaport

(Reuters Health) - Red meat – but not other types of protein - is linked to an increased stroke risk, and the odds go up the more meat people eat, a recent study suggests.

Researchers analyzed data on about 11,000 middle-aged people who didn’t have other risk factors for strokes such as diabetes or heart disease, and followed half of them for around 23 years.

The people in the study who consumed the most red meat had a 47 percent higher risk of ischemic stroke – caused by blockages in blood vessels supplying the brain - than those who typically ate the smallest amount of red meat. Protein from poultry, seafood or vegetable sources like nuts and legumes wasn’t associated with any added risk.

Even though some previous research has linked high-protein diets to strokes, the results have been mixed and the current study helps solidify the evidence suggesting that red meat in particular may pose a danger, said lead study author Dr. Bernhard Haring of the Comprehensive Heart Failure Center at the University of Wurzburg in Germany.

“It’s ok to eat red meat – preferably lean red meat – as long as you limit the amount,” Haring said by email.

To assess the link between protein consumption and stroke risk, Haring and colleagues reviewed data from diet questionnaires completed by U.S. residents aged 45 to 64 starting in 1987 and followed them through 2011 to see how many people had strokes.

The study participants were divided into five groups based on how much protein and what type they consumed. For instance, the bottom-fifth averaged about 49 grams of protein a day, representing less than 13 percent of total calories. The top-fifth averaged 93 grams of protein a day, representing 23 percent of total calories.

Compared to participants with high protein consumption, those who ate less protein on average at the start of the study were more likely to be black, current smokers and less likely to have high school diplomas or a regular exercise routine. The people who ate less protein were also less likely to be obese or take cholesterol-lowering medications.

There were no major differences in age gender, or total calories consumed among participants who ate different amounts of protein.

During a median follow-up of 22.7 years, there were 699 strokes among 11,601 participants.

The highest intake of processed meats like bacon, sausage and jerky was linked to a 24 percent higher risk of strokes, while the highest consumption of red meat was tied to a 41 percent increased risk, compared to people in the bottom-fifth for consumption of those items.

When the researchers looked just at men, the highest consumers of red and processed meats had a 62 percent higher stroke risk than men who ate the least.

Eating more eggs was linked to a 41 percent greater risk of hemorrhagic strokes, a less common type that is caused by a ruptured blood vessel in the brain. But only red meats were tied to ischemic strokes, the most common kind.

One limitation of the study is that researchers only had data on protein intake at two points in time, which the authors acknowledge might fail to account for changes in eating habits over the years.

Because the study was based on observation only and didn’t randomly assign some people to eat red meat while others abstained, it isn’t possible to determine how diet changes might help reduce the risk of future strokes, noted Dr. Jennifer Dearborn-Tomazos, a neurology researcher at Yale University School of Medicine in New Haven, Connecticut.

It’s possible, for example, that people who eat a lot of red meat also do other things that increase the risk of strokes, like not eating enough vegetables, Dearborn-Tomazos, who wasn’t involved in the study, said by email.

Even so, the study findings linking red meat to stroke risk after accounting for how much fat, carbohydrates and fiber people consumed supports traditionally held beliefs that red meat and saturated fats may increase the risk of cardiovascular disease, she said.

“This study really tells us that what we eat matters for our future cardiovascular health,” Dearborn-Tomazos said.

SOURCE: bit.ly/1MCIQ3z Stroke, online October 29, 2015.

Lifestyle changes prevent cognitive decline even in genetically susceptible individuals

I would think with any competency at all your doctor should be able to provide you lifestyle protocols to prevent cognitive decline post stroke. ROFLMAO.
https://www.alphagalileo.org/ViewItem.aspx?ItemId=183025&CultureCode=en

25 January 2018 University of Eastern Finland

Enhanced lifestyle counselling prevents cognitive decline even in people who are carriers of the APOE4 gene, a common risk factor of Alzheimer's disease, according to a new study published in JAMA Neurology.

The two-year FINGER trial involved 60-77 year-old people living in Finland and with risk factors for memory disorders. The study participants were divided into two groups: one of the groups was given regular lifestyle counselling and the other enhanced lifestyle counselling. Enhanced counselling involved nutrition counselling, physical and cognitive exercises, and support in managing the risk of cardiovascular diseases.

Earlier findings from the FINGER trial have shown that the regular lifestyle counselling group had a significantly increased risk of cognitive and functional impairment compared to the intervention group, i.e. the group receiving enhanced counselling.

Now the researchers analysed whether the presence of the APOE4 gene affected the intervention results. The analysis included 1,109 persons of whom 362 were carriers of the APOE4 gene. The findings show that enhanced lifestyle counselling prevented cognitive decline despite the presence of the risk gene. Analyses carried out within the groups also indicate that the intervention results might even be better in carriers of the APOE4 gene.

"Many people worry that genetic risk factors for dementia may thwart potential benefits from healthy lifestyle changes. We were very happy to see that this was not the case in our intervention, which was started early, before the onset of substantial cognitive impairment," says Adjunct Professor Alina Solomon, the lead author of the study.

Professor Miia Kivipelto, the principal investigator of the FINGER trial, adds: "The FINGER intervention model is now being adapted and tested globally in the World Wide FINGERS initiative. New clinical trials in diverse populations with a variety of geographical and cultural backgrounds will help us formulate global dementia prevention strategies".

The study was carried out in collaboration between the University of Eastern Finland, the Finnish National Institute for Health and Welfare, the University of Helsinki, the University of Oulu, and Karolinska Institutet in Sweden.

• Full bibliographic informationEffect of the Apolipoprotein E genotype on Cognitive Change During a Multidomain Lifestyle Intervention. A Subgroup Analysis of a Randomized Clinical trial. Solomon A, Turunen H, Ngandu T, Peltonen M, Levälahti E, Helisalmi S, Antikainen R, Bäckman L, Hänninen T, Jula A, Laatikainen T, Lehtisalo J, Lindström J, Paajanen T, Pajala S, Stigsdotter-Neely A, Strandberg T, Tuomilehto J, Soininen H, Kivipelto M. Jama Neurology. Published online January 22, 2018. doi:10.1001/jamaneurol.2017.4365

Kinect-based individualized upper extremity rehabilitation is effective and feasible for individuals with stroke using a transition from clinic to home protocol.

You'll have to read the manuscript at the link OR just ask your doctor. ROFLMAO.

Kinect-based individualized upper extremity rehabilitation is effective and feasible for individuals with stroke using a transition from clinic to home protocol.

Blueberry Vinegar Boosts Memory in Lab - mice

Not to be done for 50 years until your doctor comes up with a protocol. Who cares if you are dead by then? Certainly not you, you'll be dead. I'm sure your doctor reads the ACS’ Journal of Agricultural and Food Chemistry and will immediately recognize the importance of this and recruit researchers for human testing.  ROFLMAO
http://www.alzheimersweekly.com/2018/01/blueberry-vinegar-boosts-memory-in-lab.html

DIET & NUTRITION: Blueberry vinegar may fight dementia by improving cognitive function. Learn about improvements to short-term memory in the lab as reported in The Journal of Agricultural and Food Chemistry.

---

In the search for new ways to fight cognitive decline, scientists report in ACS’ Journal of Agricultural and Food Chemistry that blueberry vinegar might offer some help. They found that the fermented product could restore cognitive function in mice.

Recent studies have shown that the brains of people with Alzheimer’s disease, the most common form of dementia, have lower levels of the signaling compound acetylcholine and its receptors. Research has also demonstrated that blocking acetylcholine receptors disrupts learning and memory. Drugs to stop the breakdown of acetylcholine have been developed to fight dementia, but they often don’t last long in the body and can be toxic to the liver. Natural extracts could be a safer treatment option, and some animal studies suggest that these extracts can improve cognition. Additionally, fermentation can boost the bioactivity of some natural products. So Beong-Ou Lim and colleagues wanted to test whether vinegar made from blueberries, which are packed with a wide range of active compounds, might help prevent cognitive decline.

To carry out their experiment, the researchers administered blueberry vinegar to mice with induced amnesia. Measurements of molecules in their brains showed that the vinegar reduced the breakdown of acetylcholine and boosted levels of brain-derived neurotrophic factor, a protein associated with maintaining and creating healthy neurons. To test how the treatment affected cognition, the researchers analyzed the animals’ performance in mazes and an avoidance test, in which the mice would receive a low-intensity shock in one of two chambers. The treated rodents showed improved performance in both of these tests, suggesting that the fermented product improved short-term memory. Thus, although further testing is needed, the researchers say that blueberry vinegar could potentially be a promising food to help treat amnesia and cognitive decline related to aging.
SOURCE:

• Cognitive Improving Effects by Highbush Blueberry (Vaccinium crymbosum L.) Vinegar on Scopolamine-Induced Amnesia Mice Model
  Journal of Agricultural and Food Chemistry
• The authors acknowledge funding from Konkuk University. 
• Note: ACS does not conduct research, but publishes and publicizes peer-reviewed scientific studies.

Forced Use of the Paretic Leg Induced by a Constraint Force Applied to the Nonparetic Leg in Individuals Poststroke During Walking

If this truly works your doctor should have this implemented in their hospital in the next month. ROFLMAO.  And an email address to get the protocol for this. Your doctor needs to do no thinking at all to accomplish this, thus not needing to tax their two functioning neurons.
http://journals.sagepub.com/doi/abs/10.1177/1545968317740972

Show all authors

Chao-Jung Hsu, PT, PhD¹

Chao-Jung Hsu

1Shirley Ryan AbilityLab, Chicago, IL, USA
See all articles by this author

Search Google Scholar for this author

, Janis Kim, MPT¹

Janis Kim

1Shirley Ryan AbilityLab, Chicago, IL, USA
See all articles by this author

Search Google Scholar for this author

, Elliot J. Roth, MD¹²

Elliot J. Roth

1Shirley Ryan AbilityLab, Chicago, IL, USA2Northwestern University, Chicago, IL, USA
See all articles by this author

Search Google Scholar for this author

,

William Z. Rymer, MD, PhD¹²*

William Z. Rymer

1Shirley Ryan AbilityLab, Chicago, IL, USA2Northwestern University, Chicago, IL, USA
See all articles by this author

Search Google Scholar for this author

, Ming Wu, PhD¹²

Ming Wu

1Shirley Ryan AbilityLab, Chicago, IL, USA2Northwestern University, Chicago, IL, USA
See all articles by this author

Search Google Scholar for this author

...

First Published November 16, 2017 Research Article

https://doi.org/10.1177/1545968317740972

Download PDF	Article information

Article Information

Volume: 31 issue: 12, page(s): 1042-1052

Article first published online: November 16, 2017; Issue published: December 1, 2017

https://doi.org/10.1177/1545968317740972

Chao-Jung Hsu, PT, PhD¹, Janis Kim, MPT¹, Elliot J. Roth, MD^{1, 2}, William Z. Rymer, MD, PhD^{1, 2}*, Ming Wu, PhD^{1, 2}

¹Shirley Ryan AbilityLab, Chicago, IL, USA

²Northwestern University, Chicago, IL, USA

Corresponding Author: Ming Wu, Legs and Walking Lab, Shirley Ryan AbilityLab, 355 East Erie Street, 23rd Floor, Chicago, IL 60611, USA. Email: w-ming@northwestern.edu

*This author is a member of The American Society of Neurorehabilitation.

• Abstract
• Full Text
• References
• PDF

Abstract

Background. Individuals with stroke usually show reduced muscle activities of the paretic leg and asymmetrical gait pattern during walking.
Objective. To determine whether applying a resistance force to the nonparetic leg would enhance the muscle activities of the paretic leg and improve the symmetry of spatiotemporal gait parameters in individuals with poststroke hemiparesis.
Methods. Fifteen individuals with chronic poststroke hemiparesis participated in this study. A controlled resistance force was applied to the nonparetic leg using a customized cable-driven robotic system while subjects walked on a treadmill. Subjects completed 2 test sections with the resistance force applied at different phases of gait (ie, early and late swing phases) and different magnitudes (10%, 20%, and 30% of maximum voluntary contraction [MVC] of nonparetic leg hip flexors). Electromyographic (EMG) activity of the muscles of the paretic leg and spatiotemporal gait parameters were collected.
Results. Significant increases in integrated EMG of medial gastrocnemius, medial hamstrings, vastus medialis, and tibialis anterior of the paretic leg were observed when the resistance was applied during the early swing phase of the nonparetic leg, compared with baseline. Additionally, resistance with 30% of MVC induced the greatest level of muscle activity than that with 10% or 20% of MVC. The symmetry index of gait parameters also improved with resistance applied during the early swing phase.
Conclusion. Applying a controlled resistance force to the nonparetic leg during early swing phase may induce forced use on the paretic leg and improve the spatiotemporal symmetry of gait in individuals with poststroke hemiparesis.

Scientists chart how brain signals connect to neurons

Good news. Now our stroke medical professionals can use this to devise a strategy to reconnect brain signals disrupted by your stroke. I would expect an updated strategy in weeks. ROFLMAO
https://medicalxpress.com/news/2017-12-scientists-brain-neurons.html
Scientists at Johns Hopkins have used supercomputers to create an atomic scale map that tracks how the signaling chemical glutamate binds to a neuron in the brain. The findings, say the scientists, shed light on the dynamic physics of the chemical's pathway, as well as the speed of nerve cell communications.

It's long been known that brain neurons use glutamate as a way to communicate with each other. As one neuron releases glutamate, an adjacent neuron latches onto the chemical through a structure on the neuron's surface called a receptor. The glutamate-receptor connection triggers a neuron to open chemical channels that let in charged particles called ions, creating an electric spark that activates the neuron.
"All of this happens within a millisecond, and what hasn't been known is the way receptors latch onto glutamate. Our new experiments suggest that glutamate molecules need to take very particular pathways on the surface of glutamate receptors in order to fit into a pocket within the receptor," says Albert Lau, Ph.D., assistant professor of biophysics and biophysical chemistry at the Johns Hopkins University School of Medicine.
For the research, the Johns Hopkins scientists used a supercomputer called Anton, which is run by the Pittsburgh Supercomputing Center. They also worked with researchers at Humboldt University in Berlin who specialize in recording how charged particles flow between biological membranes.
A report of the experiments will be published in the Jan. 3 issue of Neuron.
To develop their model of how glutamate might connect to brain cell receptors, Lau and Johns Hopkins research fellow Alvin Yu used a computing technique called molecular dynamic simulations, which was developed by Martin Karplus, Michael Levitt and Arieh Warshel and earned them a Nobel Prize in 2013. The simulations use Sir Isaac Newton's laws of motion and a set of mathematical rules, or algorithms to assign energy functions to atoms and the substances made from those atoms.
"It takes an enormous amount of computer processing power to do these types of simulations," says Lau.
In their experiment, Yu and Lau immersed glutamate molecules and a truncated version of the glutamate receptor in a water and sodium chloride solution. The supercomputer recorded dynamics and interactions among nearly 50,000 atoms in the solution.

"There are many ways glutamate can connect with a receptor," says Lau. But some pathways are more direct than others. "The difference is like taking the faster highway route versus local roads to get to a destination."
Yu and Lau counted how frequently they saw glutamate in every position on the receptor. It turns out that glutamate spends most of its time gliding into three distinct pathways.

Seek% buffered00:00Current time00:12Volume

Simulation of how a glutamate molecule enters one of three main pathways into the pocket of a glutamate receptor. Red-tipped ends represent negative charges; blue-tipped ones are positive charges. Credit: Alvin Yu and Albert Lau

Zooming in more closely at those pathways, the scientists found that the chemical's negatively charged atoms are guided by positively charged atoms on the neuron's glutamate receptors.
"What we see is an electrostatic connection, and the path glutamate follows is determined by where the charges are," says Lau. In the world of physics, when two objects near each other have opposing electrical charges, they attract each other.
Lau says that the positively charged residues on the glutamate receptor may have evolved to shorten the time that glutamate takes to find its binding pocket.
To test this idea, Lau teamed up with scientists at Humboldt University to introduce mutations into the gene that codes for the glutamate receptor to change positively charged residues into either negatively charged or uncharged ones.
Then, they measured the resulting electrical currents to determine if there was a change in the rate of the receptor's activation in the presence of glutamate.
The results of that experiment showed that mutated glutamate receptors activated at half the speed of the normal version of the receptor.
"If, as we think is the case, communication between neurons has to happen at a particular rate for effective brain activity, then slowing down that rate means that the brain won't work as well," says Lau. "We believe that these glutamate receptors have evolved a way to speed up the binding process."
The scientists add that, in some cases, glutamate seems to be able to bind to the receptor upside down. When this happens, the glutamate receptor's pocket can't close entirely, possibly making it unable to fully open its channels to allow ions into the neuron.
Lau says that further research is needed to determine if other compounds that target the glutamate receptor, such as quisqualic acid, which is found in the seeds of some flowering plants, tread the same three pathways that glutamate tends to follow.
So far, Lau's team has focused its computer simulations only on the main binding region of the glutamate receptor. The researchers plan to study other areas of glutamate receptors exposed to glutamate.

Explore further: Scientists capture first image of major brain receptor in action

More information: Neuron (2017). DOI: 10.1016/j.neuron.2017.11.024 , http://www.cell.com/neuron/fulltext/S0896-6273(17)31077-2

Journal reference: Neuron
Provided by: Johns Hopkins University School of Medicine

Upper-extremity robot-aided rehabilitation after stroke: A comparison of the arm and wrist robots

Of course your stroke hospital is already comparing these robots and has already selected the best. ROFLMAO.
http://www.jns-journal.com/article/S0022-510X(17)32185-8/fulltext

Y.W. HsiehCorrespondence information about the author Y.W. Hsieh

,

T.Y. Shih

,

K.C. Lin

,

C.Y. Wu

DOI: http://dx.doi.org/10.1016/j.jns.2017.08.1688

• Abstract

• Full Text

Background: The use of robot-assisted therapy (RT) in stroke rehabilitation is prominently effective, but very limited studies have directly compared the effects of different robotic rehabilitation devices.

Objective: This study examined the efficacy of proximal RT by using the InMotion ARM^TM interactive therapy system (Proximal-IMT) versus distal RT by using the InMotion WRIST^TM interactive therapy system (Distal-IMT) on motor function, muscle strength, and real-life daily activities in stroke patients.

Patients and Methods/Material and Methods: A cluster-controlled trial was conducted and a total of 40 patients with stroke were enrolled. Participants received 1 of the Proximal-IMT, Distal-IMT, or control treatment (CT) for 20 training sessions. Outcome measures were the upper-extremity subscale of Fugl-Meyer Assessment (FMA-UE), Medical Research Council (MRC) Scale, Motor Activity Log (MAL), and wrist-worn activity monitors (ie, accelerometers).

Results: There were statistically significant differences on the distal FMA-UE, total MRC, distal MRC, and MAL quality of movement scores among the 3 groups (P = 0.02 to 0.05). Post hoc comparisons revealed that the Distal-IMT group improved more than the Proximal-IMT and CT groups in distal upper-limb motor function, muscle strength, and quality of movement while using the affected arm in daily tasks.

Conclusion: We found that distal robot-aided rehabilitation using the InMotion WRIST system had superior effects on distal upper-limb motor function, muscle strength, and perceived use of the affected arm during daily tasks. Further large-scale study is suggested to confirm the long-term treatment effects of arm and wrist robots.

Conceptualization of PhysioFun game: A low-cost videogame for home-based stroke rehabilitation

You'll have to ask your doctor why this one is better than all the previous ones written about and which should already be used in your stroke department. ROFLMAO
http://ieeexplore.ieee.org/abstract/document/7847787/?reload=true

View All

3

Author(s)

Tânia Ribeiro ; Ana Isabel Veloso ; Rui Costa

View All Authors

• Abstract
• Authors
• Figures
• References
• Citations
• Keywords
• Metrics
• Media

Abstract:

Nowadays stroke represents one of the most common health care problems, being the major cause of disability worldwide. The patients, stroke victims, face a series of motor disabilities that jeopardize their own autonomy as individuals in society. With the purpose of giving back autonomy to these patients, health stakeholders recommend physiotherapy sessions to restore the damages of the stroke. However, the current physiotherapy sessions are insufficient for an effective recovery due to inadequate time per session. In this paper is presented the conceptualization of a videogame aimed to support home based rehabilitation that could lead to a low-cost effective upper limb recovery, in the context of a Portuguese reality.

Published in: Technology and Innovation in Sports, Health and Wellbeing (TISHW), International Conference on

Date of Conference: 1-3 Dec. 2016

Date Added to IEEE Xplore: 09 February 2017

ISBN Information:

DOI: 10.1109/TISHW.2016.7847787

Publisher: IEEE

• Download PDF
• Download Citations
• View References
• Email
• Print
• Request Permissions
• Export to Collabratec
• Alerts

This article is only available in PDF.

Read document

Brain-computer interface based motor and cognitive rehabilitation after stroke – state of the art, opportunity, and barriers: summary of the BCI Meeting 2016 in Asilomar

See what state of the art BCI will bring to your stroke rehabilitation. ROFLMAO.
http://www.tandfonline.com/doi/abs/10.1080/2326263X.2016.1246328,

Floriana Pichiorri, Natalie Mrachacz-Kersting, Marco Molinari, Sonja Kleih, Andrea Kübler & Donatella Mattia

Pages 1-7 | Received 30 Jul 2016, Accepted 06 Oct 2016, Published online: 24 Oct 2016

• Download citation
• http://dx.doi.org/10.1080/2326263X.2016.1246328
• Crossmark

 

          

Abstract

Non-invasive electroencephalographic (EEG) based brain-computer interfaces (BCIs) are a potential tool to support neuronal plasticity after stroke in the sub-acute and even in the chronic state. A few randomized controlled trials have demonstrated the positive effect on motor rehabilitation. Recent data also indicate that BCI training may improve cognitive rehabilitation. However, important questions remain to be addressed for implementing BCI-based rehabilitation in the clinical routine. This translational effort requires an interdisciplinary approach. The current article provides an overview of a stroke rehabilitation workshop of the 6th International Brain-Computer Interface Meeting in Asilomar, Pacific Grove, USA, held from 30 May to 3 June 2016. This workshop provided an overview of the current state of the art in BCI-based motor and cognitive rehabilitation, presented BCI set-ups shown to be effective, and concluded with a discussion of translational issues and barriers.