Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Showing posts with label heat. Show all posts
Showing posts with label heat. Show all posts

Tuesday, February 24, 2015

Frequent saunas may boost survival, Finnish study suggests

How long before your hospital installs a sauna? Find out from your doctor what the frequency and heat protocol is. Do not attempt this on your own.
The readable article here:
http://news.yahoo.com/frequent-saunas-may-boost-survival-finnish-study-suggests-173148012.html
The abstract here:
http://archinte.jamanetwork.com/article.aspx?articleid=2130724

Conclusions and Relevance  Increased frequency of sauna bathing is associated with a reduced risk of SCD, CHD, CVD, and all-cause mortality. Further studies are warranted to establish the potential mechanism that links sauna bathing and cardiovascular health.

Have your doctor reconcile sauna use with this research;

Mortality rates increase due to extreme heat and cold

Up to now it has been known that high temperatures, amongst other things, can affect the blood-clotting mechanism (haemostasis) and make the blood more viscous, thereby increasing the risk of thrombosis. Furthermore, as decreasing temperatures have an impact on blood pressure, it can be assumed that there is a link between cold temperatures and the increase in cardiovascular events and stroke.



Wednesday, July 30, 2014

Mortality rates increase due to extreme heat and cold

So maybe I shouldn't go winter camping in -40F weather anymore. No worries about running in hot weather, I can't run yet.
http://www.alphagalileo.org/ViewItem.aspx?ItemId=144086&CultureCode=en
When temperatures are extremely high or low, there is a significant increase in the number of deaths caused by heart failure or stroke. This has been confirmed by epidemiological studies conducted by researchers at the Helmholtz Zentrum München, who have now published their results in the medical journal Heart.
Epidemiological studies have repeatedly shown that death rates rise in association with extremely hot weather. The heat wave in Western Europe in the summer of 2003, for example, resulted in about 22,000 extra deaths. A team of researchers led by Dr. Alex-andra Schneider at the Institute of Epidemiology II at the Helmholtz Zentrum München examined the impact of extreme temperatures on the number of deaths caused by cardiovascular disease in three Bavarian cities and included both high and low temperatures in the study.
“Our findings confirm the results of our previous studies, which indicated that the elderly and people with pre-existing medical conditions respond particularly sensitively to heat and cold,” says Alexandra Schneider. “If you are aware of the effects of air temperature on health, you can identify population subgroups who are particularly at risk and take preventive action.”
The elderly are particularly at risk

Dr. Susanne Breitner, Dr. Alexandra Schneider and Prof. Annette Peters evaluated almost 188,000 deaths due to cardiovascular disease between 1990 and 2006 in the cities of Munich, Nuremberg, and Augsburg. They were able to demonstrate that when temperatures rose from 20°C to 25°C or fell from -1°C to -8°C, the number of deaths from cardiovascular disease increased significantly by 9.5% and 7.9%, respectively. While the effects of the heat lasted for one or two days, the effects of cold weather lasted for up to two weeks. Elderly people were most affected. The impact on death rates due to heart failure, arrhythmia and stroke was particularly striking.
The mechanisms that cause these deaths, however, are not yet fully understood. Up to now it has been known that high temperatures, amongst other things, can affect the blood-clotting mechanism (haemostasis) and make the blood more viscous, thereby increasing the risk of thrombosis. Furthermore, as decreasing temperatures have an impact on blood pressure, it can be assumed that there is a link between cold temperatures and the increase in cardiovascular events and stroke.
Preventative programs

“Our findings give an indication of the diseases that are responsible for the observed link between air temperature and death rates, and thus provide a partial explanation as to why some people react more strongly to heat or cold than others and are, therefore, exposed to a greater health risk on hot or cold days,” says Alexandra Schneider. “These results are important in order to develop or adapt preventive programs and codes of practice.”
The scientists plan to conduct further research into the mechanisms that may be responsible for the health effects observed during cold and, in particular, hot temperatures. They are also interested in possible interactions with air pollutants, which are required in order to predict the effects of climate change on the health of the population, especially in cities and in major conurbations.
http://www.helmholtz-muenchen.de/en/news/press-releases/2014/press-release/article/24497/index.html
Full bibliographic informationBreitner S. et al. (2014). Short-term effects of air temperature on cause-specific cardiovascular mortality in Bavaria, Germany; Heart, 0:1–9. doi:10.1136/heartjnl-2014-305578

Monday, October 14, 2013

The Role and Dynamics of β-Catenin in Precondition Induced Neuroprotection after Traumatic Brain Injury

So how the hell are you going to test if this pre-conditioning  works in humans for both TBI and stroke?
http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0076129

Abstract

Preconditioning via heat acclimation (34°C 30 d) results in neuroprotection from traumatic brain injury due to constitutive as well as dynamic changes triggered by the trauma. Among these changes is Akt phosphorylation, which decreases apoptosis and induces HIF1α. In the present study we investigated the Akt downstream GSK3β/β -catenin pathway and focused on post injury alternations of β catenin and its impact on the cellular response in preconditioned heat acclimated mice. We found that the reduction in motor disability is accompanied with attenuation of depressive like behavior in heat acclimated mice that correlates with the GSK3β phosphorylation state. Concomitantly, a robust β catenin phosphorylation is not followed by its degradation, or by reduced nuclear accumulation. Enhanced tyrosine phosphorylation of β catenin in the injured area weakens the β catenin-N cadherin complex. Membrane β catenin is transiently reduced in heat acclimated mice and its recovery 7 days post TBI is accompanied by induction of the synaptic marker synaptophysin. We suggest a set of cellular events following traumatic brain injury in heat acclimated mice that causes β catenin to participate in cell-cell adhesion alternations rather than in Wnt signaling. These events may contribute to synaptogenesis and the improved motor and cognitive abilities seen heat acclimated mice after traumatic brain injury.

Monday, June 17, 2013

Heat as a stroke therapy

I guess I've been missing this the past 7 years. In Tampa in 100 degree weather is great. Extra sensation therapy by feeling all that sweat on your skin, including it running down your back and armpits. You can practice new movements, wiping sweat from your brow with your affected hand.  I'm surprised my therapists didn't know about this stroke protocol.
You might want to be concerned about this though;
California climate linked to ER visits The risk of heading to the ER for certain conditions, such as heart disease, diabetes, stroke, kidney disease and low blood pressure rises slightly as temperature and humidity increase, according to a new study. 
But what the hell do you think your doctor is for except to answer conundrums like this. They went to medical school, you didn't. So ask whether the better sensation therapy offsets the stroke risk.

Sunday, October 7, 2012

California climate linked to ER visits The risk of heading to the ER for certain conditions, such as heart disease, diabetes, stroke, kidney disease and low blood pressure rises slightly as temperature and humidity increase, according to a new study.

You'll have to read this carefully, maybe living in nice cold states like Minnesota and Michigan are healthier.
selected paragraphs printed here.
http://www.fyibehealthy.com/2012/10/06/study-california-climate-linked-to-er-visits/
The risk of heading to the emergency room for certain conditions, such as heart disease, diabetes, stroke, kidney disease and low blood pressure rises slightly as temperature and humidity increase, according to a new study from California.
Researchers also found that for a few conditions, including aneurysm and high blood pressure, higher temperatures were tied to a drop in ER visits.

For each 10-degree increase in temperature, they saw increased ER visits for a variety of conditions – from a 1.7 percent rise in ER visits for heart disease to a 4.3 percent rise in diabetes visits to a 12.7 percent increase in visits for low blood pressure.

The researchers speculate in their report, for instance, that a strong pattern of temperature-related visits for heart disease and ischemic stroke (the kind caused by a clot) could be a result of “increased blood viscosity… or changes in coagulation.”