Thursday, March 12, 2026
A landmark JAMA study just confirmed two physical tests that measure longevity by Super Age
My left hand grip is appalling and my occupational therapist had nothing to fix that. First I would have to pry all my fingers open before even trying to grip. My right hand is extremely strong. Curing spasticity is required first.
Omega-3 Supplementation Improves Cortico-Limbic Neuroplasticity and Reduces Amyloid-Related Pathology
Of course your incompetent? doctor and hospital didn't know about all this earlier research and DID NOTHING?
omega-3
(106 posts to February 2013)
Look at that, over a DECADE OF INCOMPETENCE and your doctor is still employed in stroke? The board of directors is so incompetent they don't know how to run an up-to-date stroke hospital!
Do you prefer your doctor and hospital incompetence NOT KNOWING? OR NOT DOING?
Omega-3 Supplementation Improves Cortico-Limbic Neuroplasticity and Reduces Amyloid-Related Pathology
* , ,
Lisa Pia Agosti
, * , *
Academic Editor: Grazyna Lietzau
Published: 04 March 2026 by MDPI in The 5th International Electronic Conference on Brain Sciences & 1st International Electronic Conference on Neurosciences session Neurodegenerative Diseases
Abstract:
Omega-3 polyunsaturated fatty acids (n-3 PUFA) are crucial for brain health and neuroplasticity. n-3 PUFA deficiency is associated with deficits in mood regulation and memory. This study evaluated the effects of chronic n-3 PUFA supplementation on the prefrontal cortex (PFC) and hippocampus (HIPP) in female rats exposed to a lifelong n-3 PUFA–deficient diet.
Female Wistar rats were exposed to either an n-3 PUFA–poor diet (rich in omega-6) or an n-3 PUFA–enriched diet (rich in α-linolenic acid) from conception until 8 weeks. After 9 weeks, n-3 PUFA supplementation was introduced until 16 weeks. At 16 weeks, neurochemical analyses were conducted in the PFC and HIPP, measuring neurotransmitters (5-HT, DA, NA), neurotrophic factors (BDNF, NGF), synaptic markers (SYN, CAMKII), and amyloid-related markers (amyloid oligomers, APP) using HPLC and Western blotting.
Our results showed that n-3 PUFA supplementation reversed most neurochemical alterations induced by the n-3 PUFA–poor diet in both the PFC and HIPP. Reduced levels of 5-HT and DA in both brain regions under the n-3 PUFA–poor diet were restored to control values following supplementation, while increased NA levels were normalized. SYN expression was reduced in both regions under the n-3 PUFA–poor diet and was restored after supplementation. CAMKII expression was also restored in the PFC, whereas no significant changes were observed in the HIPP. BDNF levels were reduced in both the PFC and HIPP under the n-3 PUFA–poor diet and were fully restored following supplementation. NGF levels were similarly restored in the HIPP, and no significant changes were detected in PFC. Amyloid-related markers showed limited recovery and remained elevated compared to control levels.
We concluded that n-3 PUFA supplementation restores neurotransmitter balance, synaptic function, and neurotrophic support, suggesting its therapeutic potential in neurodegenerative disorders. However, the partial reversal of amyloid-related markers indicates that early-life nutritional deficiency may lead to persistent neurodegenerative vulnerability.
Irisin reduces neurodegeneration and neuroinflammation in the 6-hydroxydopamine rat model of Parkinson’s disease
Didn't your competent? doctor tell you about this a long time ago? Some human research already exists; AND YOUR INCOMPETENT? DOCTOR DOESN'T KNOW ABOUT IT?
Do you prefer your doctor and hospital incompetence NOT KNOWING? OR NOT DOING?
irisin (12 posts to October 2013)
Irisin reduces neurodegeneration and neuroinflammation in the 6-hydroxydopamine rat model of Parkinson’s disease
Under a Creative Commons license
Open access
Reversing Memory Loss via the Vagus Nerve
Your competent? doctor has protocols on this already, right?
Oh, your doctor doesn't know about the vagus nerve and has nothing? Then find a better doctor.
Here's all the info on vagus nerve your doctor is very familiar with!
vagus nerve (65 posts to July 2012)
The latest here:
Reversing Memory Loss via the Vagus Nerve
Summary: Cognitive decline is often seen as a problem starting and ending in the brain, but new research suggests the “remote control” for memory is actually in the gut. Scientists found that as mice age, their gut microbiome shifts, specifically favoring a bacteria called Parabacteroides goldsteinii.
This shift triggers an inflammatory response in the intestines that silences the vagus nerve, the main communication highway to the brain. This silence effectively “numbs” the hippocampus, leading to memory loss. Remarkably, by stimulating the vagus nerve or resetting the gut microbiome, researchers turned “forgetful” old mice into sharp performers—proving that cognitive aging is not hardwired and can be reversed through the digestive tract.
Key Facts
- The Interoception Breakdown: Aging affects “interoception”—the brain’s ability to sense what is happening inside the body. A breakdown in gut-to-brain signaling is a direct driver of age-related memory decline.
- The Bacterial Culprit: The bacteria Parabacteroides goldsteinii increases with age, releasing metabolites that trigger gut inflammation and block vagus nerve activity.
- Reversing the Clock: Stimulating the vagus nerve in older mice restored their memory and spatial navigation skills to the level of 2-month-old mice, showing that “brain aging” can be modulated from the periphery.
Source: Stanford
The sight of a delectable plate of lasagna or the aroma of a holiday ham are sure to get hungry bellies rumbling in anticipation of a feast to come. But although we’ve all experienced the sensation of “eating” with our eyes and noses before food meets mouth, much less is known about the information superhighway, known as the vagus nerve, that sends signals in the opposite direction — from your gut straight to your brain.
These signals relay more than just what you’ve eaten and when you are full. A new study in mice from researchers at Stanford Medicine and the Palo Alto, California-based Arc Institute has identified a critical link between the bacteria that live in your gut and the cognitive decline that often occurs with aging.
“Although memory loss is common with age, it affects people differently and at different ages,” said Christoph Thaiss, PhD, assistant professor of pathology.
“We wanted to understand why some very old people remain cognitively sharp while other people see significant declines beginning in their 50s or 60s. What we learned is that the timeline of memory decline is not hardwired; it’s actively modulated in the body, and the gastrointestinal tract is a critical regulator of this process.”
The mouse study showed that the composition of the naturally occurring bacterial population that lives in the gut, known as the gut microbiome, changes with age — favoring some species of bacteria over others.
These changes are registered by immune cells in the gastrointestinal tract, which spark an inflammatory response that hampers the ability of the vagus nerve to signal to the hippocampus — the part of the brain responsible for memory formation and spatial navigation.
Stimulating the activity of the vagus nerve in older animals turned old, forgetful mice into whisker-sharp whizzes able to remember novel objects and escape from mazes as nimbly as their younger counterparts.
“The degree of reversibility of age-related cognitive decline in the animals just by altering gut-brain communication was a surprise,” Thaiss said. “We tend to think of memory decline as a brain-intrinsic process. But this study indicates that we can enhance memory formation and brain activity by changing the composition of the gastrointestinal tract — a kind of remote control for the brain.”
Thaiss, who is also a core investigator at Palo Alto-based Arc Institute, is a senior author of the study, which will be published on March 11 in Nature. Maayan Levy, PhD, an assistant professor of pathology and Arc Institute innovation investigator, is the other senior author. Timothy Cox, a graduate student at the University of Pennsylvania, is the lead author of the research.
“Our study emphasizes that processes in the brain can be modulated through peripheral intervention,” Levy said. “Since the gastrointestinal tract is easily accessible orally, modulating the abundance of gut microbiome metabolites is a very appealing strategy to control brain function.”
The call is coming from inside the body
The idea that hundreds of species of bacteria are nestled comfortably in our intestines used to be surprising. But the gut microbiome is experiencing a kind of media heyday as people realize that its function is critical to not just how we digest our food, but also to our overall health.
A little more than a decade ago, researchers showed that tinkering with rodents’ gut microbiomes affected the animals’ social and cognitive behaviors. Thaiss and Levy wondered whether a similar process could be responsible for the memory loss and cognitive troubles often associated with aging.
Signals from inside the body to the brain — like those that travel from the intestines to the brain via the vagus nerve — are part of what’s called interoception. In contrast, signals from outside the body, conveyed primarily by the five senses of taste, touch, smell, vision and hearing, are called exteroception.
“Exteroception is basically how we perceive the outside,” Thaiss said. “We have a lot of detailed knowledge about how this works. But we know much less about how the brain senses what is going on inside the body. We don’t know how many internal senses there are, or even all of what they are sensing. It’s clear that our exteroception capabilities decline with age — we grow to need eyeglasses and hearing aids, for example. And this study shows that aging also affects interoception.”
To test their theory that the gut microbiome plays a role in the “senior moments” many of us experience, the researchers housed young (2-month-old) mice together with old (18-month-old) mice. Living (and pooping) in close proximity exposed the young mice to the gut microbiomes of the old mice and vice versa. After one month, the researchers examined the compositions of the microbiomes of the old and young animals.
They found that the shared digs caused the microbiomes of the young mice to more closely resemble that of the older animals. When they compared the abilities of the mice to recognize a novel object, or to find the exit in a maze, the young mice with “old” microbiomes performed significantly more poorly than their peers — showing less curiosity about the unfamiliar object and bumbling about the maze in ways similar to that of old animals.
When the researchers compared young mice and old mice raised in a germ-free environment since birth (meaning neither group had gut bacteria), the young mice maintained their ability to form memories. But when they transplanted young, germ-free mice with microbiomes from old mice, the young mice again performed like older animals in the memory and cognition tests. Interestingly, the germ-free old mice did not experience a loss of memory and cognition as they aged, performing as well as 2-month-old animals.
Strikingly, treating young mice with “old” microbiomes (and, therefore, faltering cognitive abilities) with broad-spectrum antibiotics for two weeks restored the animals’ cognitive abilities, causing them to avidly investigate unfamiliar objects and scamper through the maze as well as their control peers.
“The object recognition test is like cognitive recognition tests in humans, where you are shown a series of images, then have to remember which ones you’ve seen before after some time passes,” Thaiss said.
“And the maze test is like people trying to recall where they parked their car at a large shopping center. What these tasks have in common, in mice and in people, is that they are very strongly dependent on activity in the hippocampus, because that is where memories are encoded.”
What’s different in their guts?
Digging deeper, the researchers identified specific changes that occur in the composition of the gut microbiome of mice as they age. In particular, the relative abundance of a bacteria called Parabacteroides goldsteinii increases in old mice and is directly associated with cognitive decline in the animals.
They showed that colonizing the guts of young mice with this bacterial species inhibited their performance on the object recognition and maze escape tasks, and that this deficit correlated with a reduction of activity in the hippocampus.
When they treated old mice with a molecule that activates the vagus nerve, however, the cognitive performance of the animals was indistinguishable from that of young animals.
Further experiments showed that the increasing prevalence of the Parabacteroides goldsteinii bacteria correlated with an increasing amount of metabolites called medium-chain fatty acids, and that these metabolites cause a group of immune cells in the gut called myeloid cells to initiate an inflammatory response. This inflammation inhibits the activity of the vagus nerve, the activity of the hippocampus and the ability to form lasting memories.
“The GI tract is arguably the first organ system to evolve during human evolutionary history, so the evolution of cognitive processes in the brain has undoubtedly been shaped by signals coming from the intestine,” Levy said. “It’s likely that signals from the GI tract play an important role in contextualizing memory formation.”
Thaiss added, “Basically, we’ve identified a three-step pathway toward cognitive decline that starts with gastrointestinal aging and the subsequent microbial and metabolic changes that occur.
“The myeloid cells in the GI tract sense these changes, and their inflammatory response impairs the connection between the gut and the brain via the vagus nerve. This is a direct driver of memory decline. And if we restore the activity of the vagus nerve, we can restore an old animal’s memory function to that of a young animal.”
The researchers are now investigating whether a similar gut microbiome and brain activity pathway exists in humans, and whether it also contributes to age-related cognitive decline. Importantly, vagus nerve stimulation is approved by the Food and Drug Administration as a treatment for depression or epilepsy and to aid stroke recovery. The researchers are also interested in developing ways to non-invasively monitor, and perhaps even control, the activity of peripheral neurons to affect memory formation and cognition.
“Our hope is that ultimately these findings can be translated into the clinic to combat age-related cognitive decline in people,” Thaiss said.
Researchers from Monell Chemical Senses Center in Philadelphia; the University of California, Irvine; University College Cork, Ireland; Calico Life Sciences LLC; and the Children’s Hospital of Philadelphia contributed to the work.
Funding: The study was funded by the Arc Institute, the National Institutes of Health (grants NIH DK019525, T32AG000255, F30AG081097, T32HG000046, F30AG080958, DP2-AG-067511, DP2-AG-067492, DP1-DK-140021, R01-NS-134976 and R01-DK-129691), the Burroughs Wellcome Fund, the American Cancer Society, the Pew Scholar Award, the Searle Scholar Program, the Edward Mallinckrodt Jr. Foundation, the W.W. Smith Charitable Trust, the Blavatnik Family Fellowship, the Prevent Cancer Foundation, the Polybio Research Foundation, the V Foundation, the Kathryn W. Davis Aging Brain Scholar Program, the McKnight Brain Research Foundation, the Kenneth Rainin Foundation, the IDSA Foundation and the Human Frontier Science Program.
Key Questions Answered:
Q: Can my “gut health” really determine if I have senior moments?
A: Yes! This study found that the gut acts as a regulator for the brain. When your gut bacteria shift with age, they create inflammation that “muffles” the vagus nerve. If the brain can’t hear the signals from the gut, the hippocampus (your memory center) struggles to form new memories.
Q: Is “brain aging” permanent?
A: Not necessarily. The most surprising part of the Stanford study was that cognitive decline in mice was reversible. By either stimulating the vagus nerve or clearing out “old” gut bacteria with antibiotics, the researchers were able to make old mice just as sharp and curious as young ones.
Q: Does this mean I should take probiotics for my memory?
A: While specific “memory probiotics” aren’t a thing yet, the researchers are looking at how modulating gut metabolites could control brain function. Since the gut is easily accessible, “oral interventions” to sharpen the mind are a major goal for future human clinical trials.
Editorial Notes:
- This article was edited by a Neuroscience News editor.
- Journal paper reviewed in full.
- Additional context added by our staff.
About this aging research news
Author: Krista Conger
Source: Stanford
Contact: Krista Conger – Sanford
Image: The image is credited to Neuroscience News
Original Research: Open access.
“Intestinal interoceptive dysfunction drives age-associated cognitive decline” by Timothy O. Cox, Ashwarya S. Devason, Alan de Araujo, Sydney Mason, Madhav Subramanian, Andrea F. M. Salvador, Hélène C. Descamps, Junwon Kim, Yixuan Zhu, Lev Litichevskiy, Sunhee Jung, Won-Suk Song, Adrián Cortés-Martín, Nathan T. Henderson, Kuei-Pin Huang, Thao Nguyen, Wisath Sae-Lee, Iboro C. Umana, Maria Sacta, Ryan J. Rahman, Stephen Wisser, J. Andrew D. Nelson, Ilona Golynker, Alana M. McSween, Eric F. Hohmann, Shaan Patel, Anna L. Bub, Clara Soekler, Niklas Blank, Kevt’her Hoxha, Lavinia Boccia, Andrea C. Wong, Klaas Bahnsen, Jihee Kim, Natalie Biderman, Dina Abbasian, Clarissa Shoffler, Christopher Petucci, Fiona E. McAllister, Amber L. Alhadeff, Marc V. Fuccillo, Colin Hill, Cholsoon Jang, J. Nicholas Betley, Guillaume de Lartigue, Virginia Y.-M. Lee, Maayan Levy. Nature
DOI:10.1038/s41586-026-10191-6
Optimism reduces stroke severity, inflammation
The only way to have optimism post stroke is for your competent? doctor to have EXACT 100% RECOVERY PROTOCOLS! Doesn't have them, your doctor, hospital and board of directors ARE COMPLETELY FUCKING INCOMPETENT! They have all known since medical school that stroke recovery is a complete shitshow and HAVE DONE NOTHING! In my opinion, that doing nothing to get to 100% recovery is the biggest fucking failure out there!
Optimism reduces stroke severity, inflammation
American Stroke Association International Stroke Conference – Poster Presentation WP466
Research Highlights:
- Optimistic stroke survivors had lower inflammation levels, reduced stroke severity and less initial physical disability after three months compared to less optimistic survivors, according to the findings of a small study.
- Previous studies have associated optimism with improved health outcomes for other medical conditions, however, no studies previously assessed if this association exists among stroke patient.
Embargoed until 4 a.m. CT/5 a.m. ET Wednesday, Feb. 12, 2020
DALLAS, Feb. 12, 2020 — Stroke survivors with high levels of optimism had lower inflammation levels, reduced stroke severity and less physical disability after three months, compared to those who are less optimistic, according to preliminary research presented at the Nursing Symposium of the American Stroke Association’s International Stroke Conference 2020 – Feb. 18-21 in Los Angeles. The conference is a world premier meeting for researchers and clinicians dedicated to the science of stroke and brain health.
In a small study of 49 stroke survivors, researchers examined the relationship among optimism, inflammation, stroke severity and physical disability for three months after a stroke. Researchers said that understanding how these elements relate to or impact one another may provide a scientific framework to develop new strategies for stroke recovery.
“Our results suggest that optimistic people have a better disease outcome, thus boosting morale may be an ideal way to improve mental health and recovery after a stroke,” said Yun-Ju Lai, Ph.D., M.S., R.N., the study’s first author and a postdoctoral fellow in the neurology department at The University of Texas Health Science Center at Houston.
Post-stroke inflammation is detrimental to the brain and impairs recovery. Optimism has been associated with lower inflammation levels and improved health outcomes among people with medical conditions, however, no prior studies assessed if this association exists among stroke patients.
This pilot study is a secondary analysis of data collected from a repository of neurological diseases. Outcomes included optimism levels from the revised Life Orientation Test, a standard psychological tool for measuring optimism; stroke severity evaluation through the National Institutes of Health Stroke Scale, and levels of inflammatory markers—interleukin-6 (IL-6), tumor necrosis factor alpha (TNFα) and C-reactive protein (CRP).
As optimism levels increased, stroke severity and the inflammatory markers IL-6 and CRP decreased even after considering other possible variables. However, this was not true of TNFα.
“Patients and their families should know the importance of a positive environment that could benefit the patient,” Lai said. “Mental health does affect recovery after a stroke.”
The list of study authors and disclosures are available in the abstract. The study was funded by NIH/NINDS and the American Heart Association.
Additional Resources:
- Video interview: AHA/ASA past president, Ralph L. Sacco, M.D., M.S., FAHA, offers perspective (via Skype) and researcher photo may be downloaded from the right column of the release link (click through thumbnails to select). https://newsroom.heart.org/news/optimism-reduces-stroke-severity-inflammation?preview=278409c58953cc4b3bd80e609533b04b
- Emotional & Behavioral Effects of Stroke
- Being more optimistic linked to better cardiovascular health among Hispanics/Latinos
- For more news at ASA International Stroke Conference 2020, follow us on Twitter @HeartNews #ISC20.
Statements and conclusions of study authors that are presented at American Stroke Association scientific meetings are solely those of the study authors and do not necessarily reflect Association policy or position. The Association makes no representation or warranty as to their accuracy or reliability. The Association receives funding primarily from individuals; foundations and corporations (including pharmaceutical, device manufacturers and other companies) also make donations and fund specific Association programs and events. The Association has strict policies to prevent these relationships from influencing the science content. Revenues from pharmaceutical and device corporations are available at https://www.heart.org/en/about-us/aha-financial-information.
The American Stroke Association’s International Stroke Conference (ISC) is the world’s premier meeting dedicated to the science of stroke and brain health. ISC 2020 will be held February 19-21 at the Los Angeles Convention Center in California. The 2 ½-day conference features more than 1,600 compelling scientific presentations in 21 categories that emphasize basic, clinical and translational science for health care professionals and researchers. These science and other clinical presentations will provide attendees with a better understanding of stroke and brain health to help improve prevention, treatment and outcomes for the more than 800,000 Americans who have a stroke each year. Stroke is the fifth leading cause of death and a leading cause of serious, long-term disability in the U.S. Worldwide, cerebrovascular accidents (stroke) are the second leading cause of death and the third leading cause of disability, according to the World Health Organization. Engage in the International Stroke Conference on social media via #ISC20.
Want To Reduce Inflammation? New Research Points To Optimism by mindbodygreen
The only way to have optimism post stroke is for your competent? doctor to have EXACT 100% RECOVERY PROTOCOLS! Doesn't have them, your doctor, hospital and board of directors ARE COMPLETELY FUCKING INCOMPETENT! They have all known since medical school that stroke recovery is a complete shitshow and HAVE DONE NOTHING! In my opinion, that doing nothing is the biggest fucking failure out there!
Want To Reduce Inflammation? New Research Points To Optimism
Inflammation is understood to have myriad effects on the body, some necessary, some not so much. When it comes to chronic inflammation, those effects can wreak havoc on your immune system, organs, and mental health alike. But new research says there may be a way to lower inflammation with just a thought—and an optimistic one at that.
In a study by the American Stroke Association, researchers found stroke survivors who had lower levels of inflammation and less severe strokes had something in common: more optimism.
Optimism improves disease outcome
The inflammation that can result from a stroke often impedes recovery for survivors and can go on to cause other complications. So with that in mind, researchers analyzed data from 49 stroke survivors.
They based the findings on three markers of inflammation, as well as the Life Orientation Test's standards for optimism levels. The more optimistic survivors were, the lower their stroke severity and inflammatory markers. And not only that, but after three months, survivors had less physical disability when compared to survivors who weren't as optimistic.
First author of the study Yun-Ju Lai, Ph.D., M.S., R.N., says, "Our results suggest that optimistic people have a better disease outcome, thus boosting morale may be an ideal way to improve mental health and recovery after a stroke."
Applying the findings
The team hopes this new understanding of how optimism influences stroke outcomes will offer a new approach to incorporate in stroke recovery.
Lai went on to say, "Patients and their families should know the importance of a positive environment that could benefit the patient. Mental health does affect recovery after a stroke."
Indeed, inflammation in the brain after a stroke is unavoidable as the brain tries to heal, which we know can result in impaired mental health. And additional research has shown how oppositely, pessimism can increase inflammation1.
So while these findings are particular to stroke survivors, there's sufficient evidence for all of us to stay positive.
SIH Hospitals Earn National Stroke Care Award
This is the whole problem in stroke enumerated in one word; 'care'; NOT RECOVERY!
Our non-existent stroke leadership should be demanding RECOVERY NOT 'CARE'! This is for you to solve: WSO! In my opinion, this is declaring failure a success; '1984' was not to be followed as a way to run a hospital!
My god, anyone in the business world would be fired immediately for managing or caring about something rather than delivering RESULTS. And this is why this is a complete fucking failure! This does nothing to guarantee recovery for survivors!
If your stroke medical 'professional'/hospital is touting 'care' it means they are a failure because they are delivering 'care'; NOT RECOVERY! I would never go to a failed hospital! Anytime I see the word 'care' associated with a stroke hospital; I immediately think fucking failure!
YOU have to get involved and change this failure mindset of 'care' to 100% RECOVERY! Survivors want RECOVERY, NOT 'CARE'!
I see nothing here that states going for 100% recovery! You need to create EXACT PROTOCOLS FOR THAT!
ASK SURVIVORS WHAT THEY WANT, THEY'LL NEVER RESPOND 'CARE'! This tyranny of low expectations has to be completely rooted out of any stroke conversation! I wouldn't go there because of such incompetency as not having 100% recovery protocols!
RECOVERY IS THE ONLY GOAL IN STROKE!
GET THERE!
SIH Hospitals Earn National Stroke Care Award
SIH Herrin Hospital and SIH Memorial Hospital of Carbondale recognized for fast, evidence-based stroke treatment.
Published on Mar. 11, 2026
Got story updates?Submit your updates here. ›
SIH Herrin Hospital and SIH Memorial Hospital of Carbondale have been awarded the Get With The Guidelines® – Stroke Gold Plus with Target: Stroke Honor Roll by the American Heart Association/American Stroke Association. This recognition reflects the hospitals' commitment to providing high-quality, time-sensitive stroke care(NOT RECOVERY!) that can reduce death and disability.
Why it matters
Stroke is a leading cause of death and long-term disability in the U.S. Prompt, evidence-based treatment is critical for improving patient outcomes. This award highlights SIH's dedication to coordinating emergency response and delivering rapid, safe stroke care(NOT RECOVERY!).
The details
To qualify for the award, the SIH hospitals had to meet strict standards for shortening the time between a patient's arrival and the start of life-saving treatment, such as clot-busting medication. This recognition reflects the hospitals' ability to quickly mobilize emergency physicians, nurses, imaging teams, and neuroscience specialists to provide coordinated, high-quality stroke care(NOT RECOVERY!).
- The award was announced on March 11, 2026.
The players
SIH Herrin Hospital
One of the SIH hospitals that received the national stroke care(NOT RECOVERY!)e award from the American Heart Association/American Stroke Association.
SIH Memorial Hospital of Carbondale
One of the SIH hospitals that received the national stroke care(NOT RECOVERY!) award from the American Heart Association/American Stroke Association.
American Heart Association/American Stroke Association
The organizations that awarded the Get With The Guidelines® – Stroke Gold Plus with Target: Stroke Honor Roll to the SIH hospitals in recognition of their commitment to fast, evidence-based stroke treatment.
Andrea Loggini, MD
Neurointensivist and neuroscience inpatient medical director at SIH.
Jonatan Hornik, MD
Neurointensivist at SIH.
What they’re saying
“Rapid treatment truly changes what recovery looks like for stroke patients. This recognition reflects how consistently our teams follow national guidelines and, just as importantly, how well they work together to deliver time-sensitive treatment that can help patients regain speech, movement and independence.”
— Andrea Loggini, MD, neurointensivist and neuroscience inpatient medical director at SIH (webqradio.com)
“For stroke care, every minute matters. From the moment a patient arrives in the Emergency Department, there is a coordinated response involving emergency physicians, nurses, imaging teams and the neuroscience team. That shared focus allows us to move quickly, reduce delays and give patients the best possible chance for recovery.”
— Andrea Loggini, MD, neurointensivist and neuroscience inpatient medical director at SIH (webqradio.com)
“This recognition is a direct reflection of teamwork. Not just in the Emergency Department, but starting with emergency medical services (EMS) in the field. Early identification, rapid communication and seamless handoff between EMS and our ED teams set everything in motion. Stroke care succeeds when every link in the chain works together with urgency and precision.”
— Jonatan Hornik, MD, neurointensivist at SIH (webqradio.com)
“When EMS, emergency clinicians, radiology and neuroscience teams are aligned, we can move efficiently while maintaining high standards of care. That collective effort is what allows patients to receive timely treatment and what ultimately drives better outcomes.”
— Jonatan Hornik, MD, neurointensivist at SIH (webqradio.com)
“We are incredibly pleased to recognize SIH Herrin Hospital and SIH Memorial Hospital of Carbondale for their commitment to caring for patients with stroke. Participation in Get With The Guidelines is associated with improved patient outcomes, fewer readmissions and lower mortality rates — a win for health care systems, families and communities.”
— Steven Messe, MD, volunteer chair of the American Heart Association Stroke System of Care Advisory Group (webqradio.com)
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