Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, September 30, 2026

Ethics in Neurology: Preserving Patient Autonomy in Stroke Care

 I'll ask a better question. Is it ethical to not tell your patient you KNOW NOTHING ABOUT 100% RECOVERY!

And that their treatment with tPA only fully works 12% of the time 

Ethics in Neurology: Preserving Patient Autonomy in Stroke Care

Neurologists are first introduced to the ethical principle of informed consent during medical school. They learn that informed consent is central to high-quality patient care, whether a patient is signing a consent form before surgery or discussing the risks and benefits of a treatment with their physician. This principle promotes respect for patient autonomy and ensures that physicians do not violate patients’ right to participate in decisions about their care. During medical school, neurologists may begin to see these principles as part of their emerging professional identity and recognize that, like their patients, they would not want to be excluded from important decisions about their own lives and well-being.

These principles can become harder to apply during residency, when increasing workloads leave less time for patient discussions. For some treatment decisions, it may seem easier to leave clinical uncertainty unacknowledged than to explain its complexities and nuances. A cognitively impaired patient may have limited ability to understand the information, while another patient may simply want the physician to decide. During an emergency, clinicians may have to make decisions before a full discussion is possible. With experience, informed consent becomes easier and more efficient, but some clinical situations remain challenging.

Consent for thrombolytics in acute ischemic stroke is one such situation. First, there is the time pressure. Thrombolytics are typically delivered in emergency settings, when neurons die with every passing minute. Second, physicians must quickly assess whether the patient has decision-making capacity. If not, they must identify and speak with an authorized surrogate decision-maker. The discussion requires balancing the potential benefits of treatment, including a reduction in significant disability, against the known risks for fatal or severe intracranial hemorrhage and the likelihood of no appreciable treatment effect for most patients.

The ethical challenge is balancing time-sensitive medical care with respect for patients’ values and preferences.

Although these decisions can be difficult for patients and surrogates, they remain subject to informed consent. Yet there is continued disagreement about whether informed consent is required before administering thrombolytics in these situations. Physicians have cited that tissue plasminogen activator (tPA) is FDA-approved, is the standard of care, is a medication rather than a procedure, or is an emergency treatment as justification for not obtaining consent.1 With the exception of the last example, none provides sufficient justification for bypassing informed consent.

When Can Treatment Proceed Without Consent?

The concept of “implied consent” in a medical emergency is well known, but an emergency alone does not justify excluding patients from decisions about their care. In general, foregoing informed consent is ethically justified when a patient cannot consent, no surrogate is available, and waiting for consent would cause significant harm. How long clinicians should attempt to reach a surrogate before treating without consent requires professional judgment and should be individualized to the patient’s clinical needs.

Supporting Time-Sensitive Decisions

The ethical challenge is balancing time-sensitive medical care with respect for patients’ values and preferences. Decision aids can help by incorporating shared decision-making into the informed consent process. Shared decision-making involves informing patients about medically appropriate options and their potential benefits and harms, eliciting their values and preferences, and collaborating on a treatment decision.2 For tPA in acute ischemic stroke, decision aids can help clinicians communicate absolute benefits, risks, and the possibility of no treatment effect while potentially minimizing individual clinician bias.

Despite the availability of decision tools for emergency tPA use, patients and surrogates continue to struggle with these decisions, and up to 34% of otherwise ideal candidates for reperfusion are not treated.3 Racial disparities in treatment and tPA refusal rates also contribute to worse outcomes in some populations.4 The reasons for treatment refusal are not completely understood.

One possible explanation is omission bias, in which patients and physicians overestimate the risks of providing a treatment and favor withholding it. Because risk reflects both probability and magnitude, the severity of a potential outcome such as death may carry disproportionate weight even when its probability is low.

Avoiding informed consent because of time pressure or clinical uncertainty is generally not ethically justifiable. Clinicians can instead simplify the decision-making process to accommodate time constraints and, when clinically appropriate, emphasize treatment as the default when its benefits are clear.5,6 Better decision aids may further support this process. 

Respecting patients’ choices while helping them and their surrogates navigate these decisions can uphold patient autonomy while potentially facilitating better clinical outcomes.

Pomegranate Compound Shows Potential to Improve Heart Function by 80 Percent: Study

 I bet your incompetent? doctor can't figure out that better heart function delivers more oxygenated blood to the brain and that will save more neurons in the first week. NOT DOING ANYTHING WITH THIS IS PURE INCOMPETENCE!

Your doctor does have protocols on walnuts and pomegranates already, right?


Pomegranate Compound Shows Potential to Improve Heart Function by 80 Percent: Study


Scientists Uncover Hidden Link Between Coffee, Gut and Brain Health

 If your incompetent? doctor hasn't created a 24 hour coffee station based on all this earlier research, then s/he IS NOT TRAINABLE!

How coffee protects against Parkinson’s Aug. 2014 

Coffee May Lower Your Risk of Dementia Feb. 2013

Coffee drinkers rejoice! Drinking coffee could lower the risk of Alzheimer’s disease 

And this: Coffee's Phenylindanes Fight Alzheimer's Plaque December 2018

New research suggests drinking coffee may reduce the risk of frailty May 2025

I think I'm in this category:  I never get the jitters or flushed skin.

Genetics determine how much coffee you can drink before it goes wrong

I'm doing a 12 cup pot of coffee a day with full fat milk to lessen my chances of dementia and Parkinsons. Tell me EXACTLY how much coffee to drink for that and I'll change. Yep, that is a lot more than the 400mg. suggested limit, I don't care! Preventing dementia and Parkinsons is vastly more important than whatever problems it can cause! 

Of course, your fuckingly incompetent? doctor did nothing with this from 3+ years ago! And still hasn't created a 24 hour coffee station

Dementia risk could drop by drinking just one shot daily of common beverage  August 2023 

The latest here:

Scientists Uncover Hidden Link Between Coffee, Gut and Brain Health

Popular Supplement Shows Potential to Improve Muscle Strength Without Training: Study

 What is your competent? doctor's EXACT PROTCOL  to prevent muscle loss? Doesn't have any? PURE INCOMPETENCE! 

And you haven't gotten the staff fired yet?

And doesn't know about these either?

Popular Supplement Shows Potential to Improve Muscle Strength Without Training: Study

Lower cognitive test scores among adults in their 60s may signal higher future stroke risk

 Luckily my cognition was not affected at all by my stroke at age 50.

Lower cognitive test scores among adults in their 60s may signal higher future stroke risk


Adults ages 60 to 69 who scored lower on overall thinking ability, memory and mental processing speed tests were more likely to have a future stroke, finds a study in Sweden, published in the Journal of the American Heart Association Research Highlights: People in their 60s without dementia or a previous stroke were at a higher risk of having a stroke in the future if they scored lower on overall thinking ability, memory and information-processing speed tests when compared to their peers who had better scores on the same cognitive tests. The results of a study in Sweden suggest that simple cognitive testing among sexagenarians may offer clues about future stroke risk. Embargoed until 4 a.m. CT/5 a.m. ET Wednesday, Sept. 30, 2026 DALLAS, Sept. 30, 2026 — Adults in their 60s who scored lower on tests of overall thinking ability, memory and how quickly they processed information were more likely to have a stroke years later, according to new research conducted in Sweden, published today in the Journal of the American Heart Association, an open-access, peer-reviewed journal of the American Heart Association. tudy examined the risk of first-time stroke among nearly 5,000 older adults, ages 60 to 80+, and reviewed scores of cognitive function in relation to later stroke incidence. tudy participants, all of whom had no history of dementia, stroke or transient ischemic attack (often known as a mini-stroke), completed several tests that measured overall thinking ability, memory, processing speed, verbal skills and select problem-solving skills. During an average 12 years of follow-up, more than 10% of participants had a first-time stroke. Researchers then estimated the risk of stroke for those with the lowest cognitive function scores compared to people with the highest scores.

The study found that among adults ages 60-69:

  • lower overall thinking ability was linked to an 86% higher risk of stroke;
  • lower memory scores were linked to a 68% higher stroke risk; and
  • lower information-processing speed scores were linked to a 66% higher risk of stroke.

The same cognitive-stroke risk pattern was not seen among adults ages 70-79 or age 80 and older after researchers accounted for other health and lifestyle factors.

“We chose to investigate different age spans because the older population is very diverse. For instance, while many people in their 60s may have only a few medical conditions, people in their 80s often suffer from multiple medical conditions and take numerous medications,” said lead study author Alice Askemyr, M.D., a geriatrics researcher at Lund University and Skåne University Hospital in Malmö, Sweden. “Furthermore, what serves as a risk factor in early life might not serve as one if it is acquired later in life and vice versa.“

She also noted that poor cognitive performance among the younger study participants could indicate that they may have already had an undetected brain injury. “So-called ‘silent’ brain injuries can affect both cognitive functioning and the future risk of stroke,” Askemyr said. “They affect roughly 70% of adults over age 70, so their effects may be easier to detect among people in their 60s, when such injuries and other factors that can affect cognition, including medical conditions and medications, are less common.”

Elisabeth Breese Marsh, M.D., FAHA, chair of the 2026 American Heart Association Scientific Statement, Brain Health Across the Life Span: A Framework for Future Studies, was not involved in the study and noted that, “while it is certainly important for people in their 60s, my advice would be that even people in their 40s and 50s need to be focusing on risk factor modification and habits for good overall brain health.”

Marsh is a professor of neurology and director of the Bayview Stroke Center at The Johns Hopkins University School of Medicine in Baltimore.

“This study, in conjunction with others, supports how important it is to be aggressive in managing vascular risk factors and focusing on brain health in midlife, before many people typically begin to think about it. This would improve both cognitive performance and stroke risk in older age,” she added. “However, because we don’t know for sure if there is something about the cognitive impairment itself that further increases risk of stroke, the study also provides a new potential screening factor to identify those with higher stroke risk.”

According to the American Stroke Association, a division of the American Heart Association, overall health, lifestyle and environment can affect brain health and cognition over time.

Study details, background, design and limitations:

  • The study included 4,912 adults, ages 60-80+, who enrolled in the Good Aging in Skåne project and included health data from the Swedish Board of Health and Welfare.
  • Participants were ages 60 and older (46% men) at enrollment. Data was collected from 2001 to 2023.
  • During an average follow-up period of 12.3 years, 572 participants had a stroke. Data on first-time stroke events were extracted from the Swedish National Patient Register of specialized in- and outpatient care.
  • Cognitive function was assessed using standardized tests of memory, processing speed, verbal fluency and executive function. Scores were combined and grouped by age into low, middle and high cognitive performance. Overall thinking ability was estimated as a composite of all available tests.
  • Study limitations include that the study was observational, meaning it found a link between lower cognitive test scores and stroke risk but cannot prove cause and effect.

The findings may not be generalizable to people living in other countries due to differences in healthcare, socioeconomics and lifestyle habits among countries, Askemyr noted.

Co-authors, disclosures and funding sources are listed in the manuscript.

Studies published in the American Heart Association’s scientific journals are peer-reviewed. The statements and conclusions in each manuscript are solely those of the study authors and do not necessarily reflect the Association’s policy or position. The Association makes no representation or guarantee as to their accuracy or reliability. The Association receives more than 85% of its revenue from sources other than corporations. These sources include contributions from individuals, foundations and estates, as well as investment earnings and revenue from the sale of our educational materials. Corporations (including pharmaceutical, device manufacturers and other companies) also make donations to the Association. The Association has strict policies to prevent any donations from influencing its science content and policy positions. Overall financial information is available here.

The Healthiest Carbs for Longevity, Backed by a 30-Year Study by SupervAge

 Does your competent? doctor even have a diet protocol for you? Protocol NOT a guideline!

NO? So, COMPLETELY FUCKING INCOMPETENT THEN?  

And you haven't gotten them fired yet?

The Healthiest Carbs for Longevity, Backed by a 30-Year Study

14 million views of this blog

 

I must still be interesting to some people. I have never received any direct communications from any stroke medical 'professionals'. Are they scared to talk to me?

First million took from Aug. 2010  to Sept. 2014

Second million from Sept. 2014 to May 2016

Third million from May 2016 to March 2017

Fourth million from March 2017 to November 2018

Fifth million from November 2018 to September 2021

Sixth million from September 2021 to June 2024

Seventh million was from June 2024 to January 2025. 

Eighth million was from January 2025 to June 2025

Ninth million was from June 2025 to September 2025;

Tenth million was from September 2025 to January 2026

Eleventh million was from January 2026 to April 2026

Twelfth million from April 2026 to June 2026

Thirteen million from June 2026 to July 2026

Fourteen million from July 2026 to September 2026

The first post was:



What my doctor should have told me about stroke recovery

Still has the same relevance today. And I'm still as arrogant and opinionated as ever. Arrogance is only true if you don't know what you're talking about! I do know what I'm talking about! 

Your doctor really knows nothing SPECIFIC AND EXACT to get you recovered. Ask him/her; you'll get dissembling rather than specifics! Or you'll get the craptastic saying: 'All strokes are different, all stroke recoveries are different'. In my opinion, that is the comment of a totally incompetent doctor!

Rapamycin Boosts Brain Blood Flow in Alzheimer’s Gene Carriers

 

Does your competent? doctor have enough brains to see this need and provide cerebral blood flow protocols to prevent loss of neurons during the neuronal cascade of death in the first week? Would this help that goal?

Let's see how long your doctor has been incompetent in rapamycin use.

Rapamycin Boosts Brain Blood Flow in Alzheimer’s Gene Carriers

Summary:

A pilot clinical trial from the University of Missouri reveals that low-dose rapamycin significantly boosts cerebral blood flow in healthy, middle-aged carriers of the APOE4 gene, the strongest genetic risk factor for late-onset Alzheimer’s disease. Female carriers demonstrated the most pronounced vascular improvements, highlighting the anti-aging compound’s potential as a targeted, precision preventative therapy years before cognitive decline begins.

Key Facts:

  • Selective Hemodynamic Boost: Following a four-week regimen of low-dose daily rapamycin, significant increases in cerebral blood flow were detected exclusively in APOE4 carriers, with no equivalent vascular shift observed in non-carrier controls.
  • Pronounced Response in Women: Female APOE4 carriers exhibited the greatest gains in cerebral perfusion—a critical outcome given that women account for nearly two-thirds of all diagnosed Alzheimer’s cases.
  • Early Preventative Window: The intervention targeted asymptomatic adults aged 45 to 65, establishing that vascular deficits associated with genetic Alzheimer’s risk can be therapeutically modified decades before clinical dementia symptoms appear.

Source: University of Missouri

The apolipoprotein E epsilon 4 (APOE4) allele stands as the most potent known genetic risk factor for developing sporadic, late-onset Alzheimer’s disease. Carrying a single copy triples the lifetime risk, while carrying two copies can increase that risk twelvefold.

Long before extracellular amyloid plaques or neurofibrillary tau tangles materialize, people carrying the APOE4 variant frequently exhibit subtle, insidious physiological defects—most notably, chronic hypoperfusion, or restricted cerebral blood flow to vital memory hubs.

Because healthy neurons depend on steady microvascular perfusion for oxygenation and metabolic clearance, this early vascular shortfall accelerates neural aging and lowers the threshold for neurodegenerative cascades.

Now, a clinical study led by investigators at the University of Missouri (Mizzou) suggests that this early circulatory breakdown is not an unchangeable fate. Published in the Journal of Cerebral Blood Flow & Metabolism, the findings demonstrate that the mTOR-inhibiting drug rapamycin can directly boost cerebral perfusion in cognitively healthy middle-aged APOE4 carriers.

From Preclinical Longevity to Human Precision Medicine

The research was spearheaded by Ai-Ling Lin, Ph.D., a professor in Mizzou’s School of Medicine and College of Arts and Science, and an investigator at the Roy Blunt NextGen Precision Health building.

Prior to joining Mizzou, Dr. Lin’s laboratory performed pioneering animal work showing that rapamycin—an FDA-approved immunosuppressant primarily prescribed to prevent organ transplant rejection and treat rare lung conditions—slows neurological aging and restores cerebral blood flow in transgenic APOE4 mice.

To determine whether these neuroprotective effects translate to human biology, Lin designed a clinical trial involving healthy adults aged 45 to 65 who were genetically screened for APOE4 status. Crucially, none of the participants exhibited clinical memory impairment or dementia symptoms.

Participants received a daily low dose of rapamycin over a four-week trial period. Neuroimaging assessments revealed a striking, genotype-dependent effect: only APOE4 carriers experienced significant gains in cerebral blood flow. Non-carriers taking the same regimen did not exhibit comparable vascular shifts, demonstrating that the drug acts directly upon the specific microvascular vulnerability induced by the variant.

“Alzheimer’s tends to happen more in older people, especially for those with APOE4,” said Dr. Lin. “If we can slow down aging in the brain for those people most at risk, maybe we can reduce the risk of them developing Alzheimer’s disease.”

High-Impact Protection for Women at Risk

The trial revealed another crucial clinical pattern: female APOE4 carriers derived the most dramatic improvements in brain blood flow.

This sexual dimorphism holds major clinical weight. Women represent nearly two-thirds of the global population living with Alzheimer’s disease, and female APOE4 carriers consistently exhibit more rapid cognitive deterioration and neurodegeneration than male counterparts with the same genotype.

“We also found that females with APOE4 saw the greatest improvement in brain blood flow, which is significant given that nearly two-thirds of people with Alzheimer’s are women,” Lin noted. “This research is an example of precision medicine, as we work to identify who can benefit from this drug the most.”

In addition to enhancing cerebral perfusion, the study examined systemic impacts across downstream metabolic, inflammatory, and gut microbiome pathways, suggesting that systemic longevity therapeutics can remodel whole-body physiology to support intracranial health.

Operating out of Mizzou’s Roy Blunt NextGen Precision Health facility, Lin and her colleagues plan to expand these preliminary findings into larger, longitudinal clinical trials. The overarching goal is to determine whether sustained, long-term restoration of cerebral blood flow through targeted anti-aging therapeutics can permanently delay, or even prevent, the clinical onset of Alzheimer’s disease in high-risk populations.

Editorial Notes:

  • This article was edited by a Neuroscience News editor.
  • Journal paper reviewed in full.
  • Additional context added by our staff.

About this Alzheimer’s and neuropharmacology Research:

  • Media Contact: Brian Consiglio
  • Source: University of Missouri-Columbia
  • Image Credit: Image credited to Neuroscience News
  • Original Research is Open Access: Journal of Cerebral Blood Flow & Metabolism (Sept 22, 2026). “Rapamycin increases cerebral blood flow and modulates metabolic, inflammatory, and microbiome profiles in healthy middle-aged APOE4 carriers: a pilot single-arm trial.” Authors: Chetan Aware, Caitlin Maria Neher, Carter Woods, Oleksandr Khegai, Alok Kumar Dwivedi, Maalavika Govindarajan, Kira Ivanich, Mehmet Kurt, David Beversdorf, Jianlin Cheng, Nathan Bresette, Taixing Cui, Priti Balchandani, Mitzi M. Gonzales, Aaron C. Ericsson, Talissa Altes, and Ai-Ling Lin.
  • DOI: 10.1177/0271678X261490342

Ultrasound Imaging Comparison of Crural Fascia Thickness and Muscle Stiffness in Stroke Patients with Spasticity

 Based on this, what will your competent? doctor do to cure spasticity? 

Nothing like usual? PURE INCOMPETENCE THEN! 

A fireable offence for the past two years!

Ultrasound Imaging Comparison of Crural Fascia Thickness and Muscle Stiffness in Stroke Patients with Spasticity

                                 by 1,2, 3 and 2,4,*
1
Fascia Research Institute, Myofascial Release Korea, Seoul 06136, Republic of Korea
2
Department of Physical Therapy, The Graduate School, Gachon University, Incheon 21936, Republic of Korea
3
Department and Research Institute of Rehabilitation Medicine, College of Medicine, Yonsei University, Seoul 03722, Republic of Korea
4
Department of Physical Therapy, College of Medical Science, Gachon University, Incheon 21936, Republic of Korea
*
Author to whom correspondence should be addressed.
Diagnostics 2024, 14(22), 2606; https://doi.org/10.3390/diagnostics14222606
Submission received: 13 October 2024 / Revised: 14 November 2024 / Accepted: 17 November 2024 / Published: 20 November 2024
(This article belongs to the Special Issue Advances in Ultrasound Imaging for Musculoskeletal Diseases)

Abstract

Background/Objective: 

Spasticity following stroke causes structural changes in the muscles and fascia, affecting the mobility and functional recovery of patients. Understanding these structural changes is critical to optimizing the rehabilitation strategies for patients. Therefore, in this study, we aimed to investigate the differences in crural and epimysial fascia thickness and muscle stiffness in the affected and unaffected lower limbs of chronic stroke patients with spasticity. 

Methods: 

A total of 88 patients with chronic stroke (mean age: 62.7 ± 10.2 years) were included in this study. Ankle range of motion, crural fascial thickness, and muscle stiffness in affected and unaffected lower limbs were assessed using ultrasound. Results: For the affected lower limbs, 59 patients (67.1%) exhibited a modified Ashworth scale score of 2, whereas 29 patients (32.9%) exhibited a score of 3. Ankle range of motion, fascia thickness, and muscle stiffness were also measured. The range of motion in ankle dorsiflexion and plantar flexion was significantly reduced on the affected side (p < 0.05). Crural fascia thickness was significantly greater in all regions of the affected side (anterior: 0.96 ± 0.14 vs. 0.72 ± 0.08 mm [p < 0.001]; lateral: 1.01 ± 0.14 vs. 0.75 ± 0.14 mm [p < 0.001]), and the epimysial fascia of the tibialis anterior muscle was similarly greater in the affected side (0.46 ± 0.07 vs. 0.34 ± 0.03 mm [p < 0.001]). However, no significant differences in muscle stiffness were observed between the affected and unaffected sides (p > 0.05). 

Conclusions: 

Overall, these findings revealed significant fascial thickening with only minimal changes in muscle stiffness on the affected side, highlighting the importance of controlling fascial changes for post-stroke spasticity management.

The generous act that can slow brain ageing

 Did your competent? doctor get you recovered enough to help others?

Well, I'm not really a volunteer. I have much more likely things to do to prevent dementia. 

The generous act that can slow brain ageing

We all know that we could and should be doing more to help others but with busy work lives, busy home lives and attempting to make time for self-care, there often just aren’t enough hours in the day.

However, new research from the University of Texas at Austin has revealed that, well, a bit selfishly, helping others could also be the secret to helping our own long-term brain health.

This is essential as according to Alzheimer’s Research UK, 982,000 people are estimated to be living with dementia in the UK and this number is predicted to rise to 1.4 million by 2040.

Helping others could slow down brain ageing

The study, which followed more than 300,000 adults in the US over two decades found that people who consistently helped others outside of the home showed a slower rate of age-related decline. 

This decline was reduced by 15-20% among those who either volunteered formally or helped in informal ways by doing things like helping neighbours, family or friends.

Notably, the most consistent benefit was found when people spent around two to four hours per week helping others. So, even a few hours one evening or an hour here or there could make a significant difference.

Wild.

How to get started in volunteering

Reach Volunteering offers the following advice to those that have never volunteered before: “If you’ve never volunteered before, start with a time limited project, or a short-term commitment. This will give you a chance to try out volunteering and experiment with what works for you.

 Related video:  5 daily habits doctors say can help protect your brain (KHOU-TV Houston)

 “Think carefully about what you can reasonably offer. Consider how much time you can spare, whether you can travel or if remote working would be best for you, and what sort of commitment you’re willing to make. Don’t overstretch: work out what you can confidently commit to and start there – you can always build on it later.

Science Has Found the Number of Steps That Can Keep Your Brain Healthy—And It’s Not That Many

 Did your competent? doctor get you recovered enough to do this number and at a brisk pace? 

NO, So COMPLETELY FUCKING INCOMPETENT THEN? 

Haven't you gotten that staff fired yet for incompetence? Why not? Don't believe in paying it forward? If earlier stroke survivors had demanded 100% recovery and gotten the staff to initiate the research to do that you would be 100% recovered now!

Science Has Found the Number of Steps That Can Keep Your Brain Healthy—And It’s Not That Many

And the pace you need to do it at; 15 Minutes of Walking (at This Walking Pace) Adds Years to Your Life, Study Says

DO YOU REALLY need to walk 10,000 steps a day to see results? When it comes to the brain, scientists are now saying you don’t. In fact, a study published earlier this week in Nature Medicine calculated the minimum you need to walk to lower your Alzheimer’s disease risk. Those steps may also explain why some people develop the disease at a faster rate than others.

This sheds light on why some people who appear to be on an Alzheimer’s disease trajectory don't decline as quickly as others,” says Jasmeer Chhatwal, MD, PhD, a neurologist at the Mass General Brigham department of neurology and senior study author. “Lifestyle factors appear to impact the earliest stages of Alzheimer's disease, suggesting that lifestyle changes may slow the emergence of cognitive symptoms if we act early.”

For people without a genetic predisposition to Alzheimer's, prior research estimates 45 percent of Alzheimer’s disease cases are potentially preventable. That's by making simple changes to your life, like getting regular exercise. Want to know if you’re hitting this daily move goal? Read on to know more.

What’s the magic number to walk for brain health?

Neurologists have found a daily target of 3,000 to 5,000 steps is the minimum you need to reduce Alzheimer’s risk. It’s a pretty specific number and less than you might expect when 10,000 steps is a popular step goal.

 Related video:  The daily habits research says can add ten years to your life (Talking With Docs)

 Researchers came up with the range after analyzing step count data of 296 adults between 50 to 90 years old with elevated levels of amyloid-beta, a protein that builds up in an Alzheimer’s brain. Afterward, Chhatwal and her team used PET imaging to scan each of their brains to measure baseline levels of amyloid-beta plaques and tau tangles (both hallmarks of Alzheimer's disease). Every adult was given cognitive tests each year for an average of 9 years along with repeated brain images to track changes in tau.

Daily walking of 3,000 to 5,000 steps was linked to a 3-year delay in cognitive decline. And as a lot of wellness research has pointed to—the more you walk, the better it is for your body. The brain is no exception.

For people who regularly walked 5,001 to 7,500 steps a day, cognitive decline was delayed by 7 years compared to people who walked less. The people who had the most rapid decline in cognition and daily functioning were people who live a sedentary lifestyle (walking fewer than 5,000 steps per day).


How does daily walking protect against Alzheimer’s?

There are a lot of ways that walking helps the brain, explains Vernon Williams, MD, sports neurologist and founding director of the Center for Sports Neurology and Pain Medicine at Cedars-Sinai Orthopaedics. For example, regular physical activity like walking stimulates the release of endorphins, which are natural mood elevators with anti-inflammatory properties.

Exercise is also linked to promoting neuronal growth and greater blood flow to the brain, adds Nikhil Palekar, MD, director of the Stony Brook Center of Excellence for Alzheimer's Disease at Stony Brook Medicine. “Regular walking also improves metabolic health, as it helps reduce weight, cholesterol, and blood sugar levels, all of which are known risk factors for dementia in older age."

The current study shows that walking also helps in another way: slowing down tau buildup. People who regularly walked and showed slower cognitive decline had tau proteins accumulate more slowly compared to sedentary adults.

We want to empower people to protect their brain and cognitive health by keeping physically active,” says Wai-Ying Wendy Yau, MD, a cognitive neurologist in the Mass General Brigham department of neurology and lead study author. "Every step counts—and even small increases in daily activities can build over time to create sustained changes in habit and health.” Ultimately, they hope 3,000 to 5,000 steps is less daunting than tackling a 10,000 step count and will encourage people to start moving sooner.


One common midlife condition linked to 42% higher dementia risk

 Your competent? doctor is RESPONSIBLE  for your 100% recovery so you maintain your muscle strength. 

CANT DO THAT; PURE INCOMPETENCE!

One common midlife condition linked to 42% higher dementia risk

I was in excellent shape at 50 when the stroke occurred, biking 4 miles each way commuting to work 9 months of the year in Minneapolis. March-November, sometimes biking home in the snow, had to put a rain cap on my helmet to stop the cold air from blowing thru the vents in the helmet onto my bald head.

3 years post stroke at a physical I had a resting heart rate of 54 at age 53, level of an athlete. My doctor asked what exercises I was doing; 'I've done no exercises for the past 3 years'.  And now 20 years past the stroke my fitness has declined a bit, ALL BECAUSE MY STROKE MEDICAL 'PROFESSIONALS' COMPLETELY FAILED AT GETTING ME 100% RECOVERED!

A common midlife condition affecting many older adults could significantly raise an individual’s risk of dementia, according to new research.

Published in International Journal of Food Sciences and Nutrition, the study found that maintaining muscle strength as a person navigates midlife is crucial in the fight against dementia when they age.

An important warning sign’

That finding is somewhat complicated, since another major risk factor for dementia is obesity. If a person loses weight without strength training, they are likely to lose muscle as well.

Counterintuitively, that could increase their dementia risk, even as their body fat goes down.

“It’s not just about how much you weigh—maintaining muscle strength and function also appears to be important as we age,” lead researcher Dr. Uraiporn Booranasuksakul from Burapha University said in a press release.

“Our findings suggest losing muscle could be an important warning sign for dementia risk.”The researchers found that sarcopenia—the medical term for loss of strength and muscle—was associated with a 42-percent greater risk of dementia. Sarcopenia often occurs when a person gets older.

Interestingly, the researchers found that obesity before the age of 65 was linked to a nine-percent greater risk of dementia.

However, after an individual turns 65, obesity was linked to a 17-percent lower risk—a finding that showed how crucial strength is as a person ages.

“We found that obesity in middle age was linked to a higher risk of dementia, but this relationship changed in later life,” study corresponding author Mario Siervo said in the release.

“This does not mean people should gain weight as they get older. Rather, it shows us that the relationship between body weight, aging and dementia is complex.”

‘A lot we don’t know’

Siervo added that the relationship between weight loss, muscle loss and dementia is complex, and more research is needed.

“There is still a lot we don’t know about how changes in muscle and body fat interact over time,” Siervo said.

The research is especially relevant as weight-loss drugs like Ozempic and Mounjaro sweep through the market.

Studies linking weight loss to lower risk of dementia suggest that GLP-1s could be beneficial for an aging population, especially in the United States where the CDC says two in five adults experience obesity.

Other research shows that almost half of dementia cases could be prevented by lifestyle changes.

Still, the latest study suggests that weight loss alone doesn’t tell the whole story.

“Maintaining muscle is an important part of healthy aging, and regular physical activity and good nutrition can help support muscle strength and function,” Booranasuksakul said.

Newsweek has reached out to the study authors for comment via email.

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New study suggests the aging brain is adapting, not declining

 My occupation for 38 years was a mainframe programmer in Assembler and COBOL; damned difficult problems were solved. My social connections are extensive. My aging brain at 70 is working great!

New study suggests the aging brain is adapting, not declining

 Have you ever walked into a room and forgotten why you’re there? Or struggled to recall where you left your keys? These memory lapses aren’t unusual, especially as we age, but a new paper published this month in the journal Perspectives on Psychological Science suggests there may be an overlooked biological reason for it. 

A call for more research

 “I think it’s provocative,” said Patricia Reuter-Lorenz, a professor of psychology and neuroscience at the University of Michigan who conducted some of the studies cited in the new paper. “It’s taking a new step that’s building on ideas that have been in the literature on aging for several decades, including the idea that aging involves adaptive processes,” she said. Reuter-Lorenz’s work has pointed to older adults using their frontal cortex to compensate for the weakening of the hippocampus. But she said she is interested in the silver linings of old age and is currently studying emotion later in life. She said her work is “trying to understand the tendency for older adults to have a more positive outlook and better emotion regulation.” It is advancing existing research showing that older adults tend to focus more on the positive aspects of a situation, and remember them more than negative ones. Both Reuter-Lorenz and Fernandez say the idea of brain aging as an adaptive phase more than a declining one needs more study. 

How to help your brain age well

Regardless of whether scientists believe that the older brain is adapting to a new phase of life or compensating for decline, they suggest that there are ways we can all age better. In Fernandez’s paper, he and his colleagues write that older people who “engage in lifelong learning,

complex occupations

, or

high social activity

” tend to have more flexible brains. That means they can continue to navigate complicated tasks even as the hippocampus shrinks. Reuter-Lorenz agreed that

lifestyle factors are key

. “Solid fitness, a good diet and sleep are all very important. And maintaining an active social network and cognitive engagement,” she said. “Those are the pillars.”Reuter-Lorenz also emphasized that the upsides of aging should continue to be acknowledged by researchers, especially since having a positive view of aging seems to help people age better.“There are treasures in aging that can be cherished,” Reuter-Lorenz said. “And that may only come with age.”