Deans' stroke musings: osteopath

Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Showing posts with label osteopath. Show all posts

Saturday, December 31, 2011

Effect of Cranial Osteopathic Manipulative Medicine on Cerebral Tissue Oxygenation

Are we supposed to believe that someone can manipulate our skull enough to create waves in our cerebral spinal fluid? And that that will help us? I think this is what Hercules(Kevin Sorbo) thought helped his recovery.
http://www.jaoa.org/content/111/12/660.abstract

Abstract

Context: The use of cranial osteopathic manipulative medicine (OMM) to alter cerebral tissue oxygen saturation could play a role in the maintenance of cerebral homeostasis.

Objective: To examine the effects of cranial OMM on cerebral tissue oxygen saturation (S_CTO₂) and cardiac autonomic function in healthy adults.

Methods: Cranial OMM augmentation and suppression techniques and sham therapy were randomly applied to healthy adults. During cranial OMM and sham therapy, S_CTO₂ of the prefrontal cortex was determined bilaterally by using near-infrared spectroscopy. Heart rate, blood pressure, and systemic arterial blood oxygen saturation (SaO₂) were also measured. Power spectral analysis was applied to continuous 4-minute R-R intervals. Measurements were made during 2-minute baseline periods, during 4-minute applications of the techniques, and during 5-minute recovery periods.

Results: Twenty-one adults (age range, 23-32 y) participated in the present study. Differences in mean baseline measurements for the augmentation technique, suppression technique, and sham therapy were not statistically significant for heart rate, blood pressure, SaO₂, left S_CTO₂, or right S_CTO₂. During the suppression technique, there was a statistically significant decrease in both left (slope [standard deviation]= -0.33 [0.08] %/min, R²=0.85, P=.026) and right (slope [standard deviation]=-0.37 [0.06] %/min, R²=0.94, P=.007) S_CTO₂ with increased cranial OMM time. However, neither the augmentation technique nor the sham therapy had a statistically significant effect on S_CTO₂. Decreases in normalized low-frequency power of R-R interval variability and enhancements of its high-frequency power were statistically significant (P=.05) during cranial OMM and sham therapy, indicating a decrease in cardiac sympathetic influence and an enhanced parasympathetic modulation.

Conclusion: The cranial OMM suppression technique effectively and progressively reduced S_CTO₂ in both prefrontal lobes with the treatment time.