Vitamin D Blog: A Bedroom Boost

This is just too good to pass up. I wish this was one of those things I might need to be concerned about but right now I don't know. It might be a convenient excuse to get your older male relatives to take it.
What do you think is more important to men? Preventing demntia or ED?

The Vitamin Which May Reduce Risk of Alzheimer’s and Dementia 

 Vitamin D Blog: A Bedroom Boost

Hypertension: The Link Between Erectile Dysfunction and Coronary Artery Disease

I just liked this because my solution to this problem is beer.
Follow the thought process;
endothelial function was significantly improved only after beer consumption
Ergo, beer will solve this. You can thank me after seeing what your doctors' pontification is.

http://online.liebertpub.com/doi/full/10.1089/jomh.2013.1503

Jacob Rajfer, MD,¹ and Martin M. Miner, MD^2,3

¹Department of Urology, UCLA School of Medicine, Los Angeles, California.

²Department of Family Medicine and Department of Urology, Warren Alpert School of Medicine, Brown University, Providence, Rhode Island.

³Men's Health Center, The Miriam Hospital, Providence, Rhode Island.

Address correspondence to:

Martin M. Miner, MD

The Warren Alpert School of Medicine of Brown University

164 Summit Avenue

Providence, RI 02906

E-mail: Martin_Miner@Brown.edu

ABSTRACT

The ability to attain and then maintain an erection is the result of a dynamic balance between arterial inflow and cavernosal outflow. Any imbalance that either decreases arterial inflow and/or increases cavernosal outflow may result in symptomatic erectile dysfunction (ED). Current data suggest that vasculogenic ED is of arterial origin and primarily the result of the development of systemic endothelial dysfunction (EnD). Since systemic EnD is heralded as an early marker of cardiovascular disease, specifically coronary artery atherosclerotic heart disease (CAD), it has been suggested that the onset of ED can be used as a marker for either the presence, or future development, of CAD. However, the failure to find arterial dysfunction in some young men with largely vasculogenic ED, combined with the recognition that most men with ED, regardless of age, appear to have at least some cavernosal smooth muscle dysfunction, has prompted a reexamination of the roles of vascular endothelium and the cavernosal smooth muscle in the development of aging-related ED, as well as a reexamination of the significance of ED as a marker of subsequent cardiovascular disease. The unifying concept that explains all these events is the aging-related apoptosis that occurs within vascular smooth muscle. In the penis, this smooth muscle apoptosis will cause cavernosal veno-occlusive dysfunction (CVOD), which initially affects a man's refractory period. In the peripheral vascular system, the apoptotic process leads to an increase in peripheral vascular resistance, and patients may present with hypertension. Hypertension and CVOD, then, represent the same pathophysiologic disorder in two different organ systems.