New study suggests unexpected link between long sleep and Alzheimer’s

 But do you have cause and effect correct? Early undetected Alzheimers may cause long sleep!

New study suggests unexpected link between long sleep and Alzheimer’s

Researchers found that older adults who routinely sleep 10 hours or more a night had elevated levels of a blood protein considered an early warning sign of dementia.

Sleeping long hours every night may be linked to elevated levels of a blood protein widely regarded as a telltale sign of Alzheimer’s disease, according to a new study by the University of Texas at San Antonio’s academic health center.

UT Health San Antonio reports that, among the 2,410 participants, those who slept 8.5 to nine hours a night had higher levels of phosphorylated tau 181, or p-tau181, a modified form of tau protein associated with cognitive decline. Researchers said p-tau181 levels increased “most sharply” among sleepers who slept more than 10 hours a night. “A lot of people worry about whether their sleep habits are affecting their brain health,” said Vanessa M. Young, PhD, MS, a postdoctoral research fellow at the center’s Glenn Biggs Institute for Alzheimer’s and Neurodegenerative Diseases. “Because this is a snapshot in time rather than a long-term study, we cannot say that long sleep causes Alzheimer’s, but the findings suggest it may be worth monitoring, and that more sleep is not always better for brain health.”

Participants had an average age of 70. Just over 55 percent were female.

Related video: Alzheimer's disease study (KARE-TV Minneapolis St. Paul)The study follows up onby UT Health San Antonio that found sleeping nine or more hours a night was associated with worse cognitive performance, especially among people diagnosed with depression. The latest study, conducted by some of the same researchers, was more extensive. It accounted for multiple health factors and examined blood-based markers that have been tied to Alzheimer’s and neurodegeneration in relation to participants’ self-reported sleep hours. “Sleep is a promising modifiable risk factor linked to the disease,” researchers said, “but existing evidence has remained limited and inconclusive.” people diagnosed with depression. The latest study, conducted by some of the same researchers, was more extensive. It accounted for multiple health factors and examined blood-based markers that have been tied to Alzheimer’s and neurodegeneration in relation to participants’ self-reported sleep hours. “Sleep is a promising modifiable risk factor linked to the disease,” researchers said, “but existing evidence has remained limited and inconclusive.”

Three other proteins associated with brain cell damage were also tested but, unlike p-tau181, they appeared to have no association with sleep duration, researchers said.

While the study found elevated levels of p-tau181 among habitual long sleepers, UT Health San Antonio said more research is needed to determine whether the association can ultimately be tied to Alzheimer’s risk.

The center’s findings coincide with a study published last week in JAMA that said simple blood tests may be able to predict who will develop cognitive impairment up to a decade before the first symptoms surface. The most accurate test measured levels of a mutation of a different tau protein, phosphorylated tau 217 (p-tau217).

The study of nearly 2,700 older adults on three continents found that people with high levels of the p-tau217 biomarker had a 38 percent chance of developing cognitive impairment within five years and a 78 percent chance within 10 years.

Further research and clinical trials could lead to the development of new therapies that may one day slow or even stop Alzheimer’s progression, researchers said.

An estimated 57 million people worldwide are affected by dementia, the San Antonio study noted, with Alzheimer’s diagnosed in 60 to 70 percent of all cases.

“Even with recent advances in disease-modifying therapies, Alzheimer’s remains a profound medical and societal challenge,” researchers said.

Young suggested that long sleepers might want to bring up the new study’s findings during their next doctor visit. “In plain terms, if you regularly find yourself sleeping nine to 10 hours or more a night,” she said, “it may be worth mentioning to your doctor as a useful conversation starter about your sleep quality and overall brain health.”

This post originally appeared at inc.com.

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Introducing the EASE Trial: A New Hope for Stroke Survivors Living with Emotionalism

Why are you treating depression rather than preventing it with 100% recovery protocols? You're that blitheringly stupid, you don't understand cause and effect?

PLC(Pathological laughing and crying) is a common emotional consequence of stroke. Approximately 15% to 20% of patients may experience this condition during the first year after stroke.

More commonly known as emotional lability.

Introducing the EASE Trial: A New Hope for Stroke Survivors Living with Emotionalism

Professor Niall Broomfield

EASE Lead Investigator

Introducing the EASE Trial (Evaluating Antidepressants for Emotionalism after Stroke), a groundbreaking research initiative aimed at improving the lives of stroke survivors experiencing emotionalism. This trial is now active in several areas of the UK.

What is Post-Stroke Emotionalism

Emotionalism

refers to a condition characterised by excessive or uncontrollable emotional responses. In the context of stroke survivors, emotionalism often manifests as sudden and inappropriate episodes of crying or laughing that are difficult to control. This condition can significantly impact the quality of life, leading to distress, embarrassment, and social withdrawal.The EASE Trial is a multi-centre, randomised, double-blind, placebo-controlled study designed to evaluate the effectiveness of Sertraline (50 mg once daily for six months) in treating emotionalism in individuals who have experienced a stroke.

The EASE Trial aims to investigate whether the antidepressant Sertraline can effectively treat this condition in stroke survivors.

The trial is currently opening more UK sites each month and is already recruiting participants at stroke clinics at the following hospitals;

• Norfolk and Norwich University Hospitals NHS Foundation Trust (Norfolk and Norwich University Hospital)
• NHS Lanarkshire (University Hospital Hairmyres)
• Torbay and South Devon NHS Foundation Trust (Torbay Hospital)
• Lewisham and Greenwich NHS Trust (University Hospital Lewisham)
• St George’s University Hospitals NHS Foundation Trust (St Georges Hospital)
• Somerset NHS Foundation Trust (Musgrove Park Hospital)
• Royal Wolverhampton NHS Trust (New Cross Hospital)
• Cambridge University Hospitals NHS Foundation Trust (Addenbrookes)
• Newcastle upon Tyne Hospitals NHS Foundation Trust (Royal Victoria Infirmary/Freeman Hospital)
• Liverpool University Foundation Trust (Aintree University Hospital)
• Northern Care Alliance (Northern Care Alliance)
• Maidstone and Tunbridge Wells NHS Trust (Maidstone Hospital)
• Bedfordshire Hospitals NHS Foundation Trust (Luton & Dunstable University Hospital)
• Wirral University Teaching Hospitals NHS Trust (Arrowe Park Hospital)
• Royal Stoke University Hospital  (University Hospitals of North Midlands NHS Trust)
Milton Keynes University Hospital (Milton Keynes University Hospital NHS Foundation Trust)

Individuals over the age of 18 who have experienced a stroke and exhibit symptoms of emotionalism are eligible to participate. Participation is entirely voluntary, and individuals can withdraw at any time without affecting their standard medical care.

What Does Participation Involve?

Participants will undergo a series of steps, including:Registering Interest: Completing a consent form to be contacted for eligibility checks.Testing for Emotionalism: A short assessment to confirm symptoms.

Eligibility and Safety Checks: Conducted by a clinical professional.

Consent and Baseline Questionnaires: Completing consent forms and initial questionnaires.

Taking Trial Medication: Randomly receiving either Sertraline or a placebo for six months, followed by a reduced dose for one month.

Follow-Up Questionnaires: Completing questionnaires at 3, 6, and 12 months to monitor progress.

For more general information on the trial, please visit 

https://norwichctu.uea.ac.uk/ease/   

Prognostic Value of Adding Magnetic Resonance Imaging to Computed Tomography in Acute Ischemic Stroke

 What stupidity, an additional scan does not lead to better recovery! You don't understand cause and effect at all!

Prognostic Value of Adding Magnetic Resonance Imaging to Computed Tomography in Acute Ischemic Stroke

Kaixiang Chen, Jiafeng Ni, Bo Yin

First published: 15 April 2025

https://doi.org/10.1002/jcu.23998

Funding: The authors received no specific funding for this work.

Kaixiang Chen and Jiafeng Ni contributed equally to this study.

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ABSTRACT

Objective

To assess if magnetic resonance imaging (MRI) provides additional benefits over computed tomography (CT) in patients with acute ischemic stroke (AIS).

Methods

We retrospectively reviewed adult AIS patients who underwent an initial CT scan and received intravenous thrombolysis using rt-PA, dividing them into two groups: MRI plus CT and CT alone. Propensity-score matching (PSM) analysis was employed to reduce confounding biases.

Results

After PSM, two matched groups (168 pairs, n = 336 patients) were generated. There were no significant differences in the modified Rankin Scale (mRS) scores of 0–2 or 0–1 at 3 months between the two groups (both p > 0.05). Patients in the MRI plus CT group had significantly lower incidence rates of 7-day mortality (3.0% vs. 8.9%, p = 0.04), 30-day mortality (11.3% vs. 21.4%, p = 0.02), and symptomatic intracranial hemorrhage (SICH, 11.9% vs. 23.2%, p = 0.01). Multivariate logistic regression showed that the MRI plus CT-based regimen significantly reduced the risks of 7-day (OR = 0.02, 95% CI: 0.01–0.18; p < 0.01) and 30-day mortality (OR = 0.03, 95% CI: 0.01–0.13; p < 0.01), as well as SICH (OR = 0.27, 95% CI: 0.09–0.76; p = 0.01).

Conclusion

The addition of MRI to CT enhances prognostic value in AIS patients, as it is associated with significantly reduced risks of mortality and SICH.

Identifying Post-Stroke Depression in Patients with Aphasia: A Program Plan for Evidence-Based Screening

 Why are you working on this secondary problem when you should solve the primary problem of 100% recovery? Then this problem goes away. Do you not understand cause and effect?

Identifying Post-Stroke Depression in Patients with Aphasia: A Program Plan for Evidence-Based Screening 

This interactive, gripping presentation shines a spotlight on a vulnerable population who lacks the voice to advocate for themselves: aphasic stroke survivors. We will discuss the high prevalence of depression in aphasic patients and the necessity of utilizing non-language-based tools in the screening of post-stroke depression in this neglected population.

Presenter:  Jack Y. Lagrone, DNP, PMHNP-BC

Disclosures:  The APNA planners and faculty have no relevant financial relationships to disclose. Off-label uses will not be discussed during this presentation.

Session Length:  30 minutes

Target Audience:  RN, APRN

Learning Outcome:
Upon completion of this presentation, the participant will be able to:

• Identify the best practice screening method for identifying post-stroke depression in patients with linguistic deficits of aphasia.

Efficacy and safety of very early rehabilitation for acute ischemic stroke: a systematic review and meta-analysis

 

My god, the ABSOLUTE FUCKING STUPIDITY DISPLAYED HERE! Very early rehabilitation does nothing towards the mortality risk! You are totally missing not stopping the 5 causes of the neuronal cascade of death in the first week thus not saving millions to billions of neurons.  Those extra dead neurons are likely the reason for the increased mortality.  Do you not understand cause and effect?

Efficacy and safety of very early rehabilitation for acute ischemic stroke: a systematic review and meta-analysis

Ying Lou^1,2Zhongshuo Liu^1,2Yingxiao Ji^1,2,3Jinming Cheng^1,2,3Congying Zhao^1,2,3Litao Li^1,2,3*

• ¹Department of Neurology, Hebei General Hospital, Shijiazhuang, Hebei, China
• ²Graduate School of Hebei Medical University, Shijiazhuang, Hebei, China
• ³Hebei Provincial Key Laboratory of Cerebral Networks and Cognitive Disorders, Shijiazhuang, Hebei, China

Background: Early rehabilitation after acute ischemic stroke (AIS) contributes to functional recovery. However, the optimal time for starting rehabilitation remains a topic of ongoing investigation. This article aims to shed light on the safety and efficacy of very early rehabilitation (VER) initiated within 48 h of stroke onset.

Methods: A systematic search in PubMed, Embase, Cochrane Library, and Web of Science databases was conducted from inception to January 20, 2024. Relevant literature on VER in patients with AIS was reviewed and the data related to favorable and adverse clinical outcomes were collected for meta-analysis. Subgroup analysis was conducted at different time points, namely at discharge and at three and 12 months. Statistical analyses were performed with the help of the Meta Package in STATA Version 15.0.

Results: A total of 14 randomized controlled trial (RCT) studies and 3,039 participants were included in the analysis. VER demonstrated a significant association with mortality [risk ratio (RR) = 1.27, 95% confidence interval (CI) (1.00, 1.61)], ability of daily living [weighted mean difference (WMD) = 6.90, 95% CI (0.22, 13.57)], and limb motor function [WMD = 5.02, 95% CI (1.63, 8.40)]. However, no significant difference was observed between the VER group and the control group in adverse events [RR = 0.89, 95% CI (0.79, 1.01)], severity of stroke [WMD = 0.52, 95% CI (−0.04, 1.08)], degree of disability [RR = 1.06, 95% CI (0.93, 1.20)], or recovery of walking [RR = 0.98, 95% CI (0.94, 1.03)] after stroke. Subgroup analysis revealed that VER reduced the risk of adverse events in the late stage (at three and 12 months) [RR = 0.86, 95% CI (0.74, 0.99)] and degree of disability at 12 months [RR = 1.28, 95% CI (1.03, 1.60)], and improved daily living ability at 3 months [WMD = 4.26, 95% CI (0.17, 8.35)], while increasing severity of stroke during hospitalization [WMD = 0.81, 95% CI (0.01, 1.61)].

Conclusion: VER improves activities of daily living (ADLs) and lowers the incidence of long-term complications in stroke survivors. However, premature or overly intense rehabilitation may increase mortality in patients with AIS during the acute phase. PROSPERO registration number: CRD42024508180.

Systematic review registration: This systematic review was registered with PROSPERO (https://www.crd.york.ac.uk/PROSPERO/). PROSPERO registration number: CRD42024508180.

1 Introduction

Acute ischemic stroke (AIS) refers to the abrupt onset of focal neurological dysfunction resulting from insufficient blood supply to the brain or determined according to objective evidence of vascular origin observed through imaging or pathological examination (1). It features high incidence, recurrence, disability, and mortality worldwide (2), and represents approximately 80% of all stroke cases (3). In the Trial of Org 10,172 in Acute Stroke Treatment (TOAST) classification system, large-artery atherosclerosis and cardioembolism are the main etiologies of stroke, with contributing risk factors including cardiovascular, endocrine, and others. Stroke, as the second leading cause of death and disability worldwide according to the Global Burden of Disease Study in 2016 (4), imposes substantial health and economic burdens in both developed and developing nations. Moreover, there has been a gradual increase in stroke incidence among young populations (5, 6). The progression of ischemic stroke is commonly categorized into acute, subacute, and chronic phases; however, the temporal boundaries of these stages are inconsistently defined. In the present study, acute stroke was defined as a stroke that occurs within 7 days after the onset, subacute stroke was a stroke occurring more than 7 days and less than 3 months after the onset, and chronic stroke generally referred to a non-recurrent stroke that lasts 3 months. Despite advancements in stroke unit management and early revascularization which promote timely recovery of brain blood flow in recent years, 50% of patients became chronically disabled with low life quality (7), because neural restoration was constrained by a narrow therapeutic window and irreversible damage to neuron. Some stroke survivors experience lingering complications and sequelae, particularly motor impairment and cognitive decline (8). In a recent study, it was demonstrated that acute or subacute stroke patients with Clostridium difficile infection exhibited significant improvement in basic living ability at discharge after 3 h of daily neurorehabilitation, but no significant difference was found in comparison to non-infected patients (9). Therefore, in addition to standard care, systematic, regular and intensive rehabilitation is of great importance in the early period of stroke even in the presence of other complications such as infections, unless patients have malaise or worse symptoms.

Post-stroke rehabilitation, as a long and relatively safe intervention, is conducive to restoring limb motivation, improving walking and balancing abilities, and reducing the incidence of disability, falls and cardiorespiratory diseases (10). Initiating rehabilitation promptly after the stabilization of vital signs would help to accelerate the recovery of central nervous system and prevent potential complications (11). Sun et al. suggested that early rehabilitation could influence the expression of serum inflammatory factors, such as vascular endothelial growth factor (VEGF), tumor necrosis factor-α (TNF-α), interleukin-10, and stromal cell-derived factor-1α, and motivate endothelial progenitor cells (12), thereby promoting endothelial formation and vascular regeneration in AIS (13). However, the optimal timing for commencing early rehabilitation after stroke remains controversial, with uncertainty regarding the safety and efficacy of very early rehabilitation (VER) in patients with AIS. Firstly, for patients with post-stroke paralysis, very early out-of-bed activities may precipitate falls due to weak limb strength or poor balancing ability. Moreover, significant head position change after stroke would decrease cerebral blood flow (14), which could aggravate ischemia in the infarct area and lead to deterioration of the disease, while maintaining a supine position could increase cerebral perfusion pressure and boost collateral circulation to support the ischemic penumbra (15, 16). Despite the absence of definitive evidence and a lack of consensus regarding the optimal rehabilitation strategy, which involves starting time, frequency and intensity (17, 18), VER has been advocated within some published stroke guidelines (19, 20), and merits further exploration. Notably, a recent meta-analysis of randomized controlled trials (RCTs) conducted in 2021 revealed positive efficacy of early rehabilitation at 3 months. No statistical difference in adverse events and disability rate was noted between the VER group and control group, but the study did not assess outcomes in different endpoints (21).

This meta-analysis included RCTs to evaluate the effects of initiating VER within 48 h of stroke onset on short- and long-term recovery. Additionally, a subgroup analysis at different time points (at discharge, 3 months and 12 months) was performed to observe the dynamic changes of the efficacy and safety of VER, which could serve as a reference for clinical practice.

More at link.

Efficacy and safety of very early rehabilitation for acute ischemic stroke: a systematic review and meta-analysis

 My god, the ABSOLUTE FUCKING STUPIDITY DISPLAYED HERE! Very early rehabilitation does nothing towards the mortality risk! You are totally missing not stopping the 5 causes of the neuronal cascade of death in the first week thus not saving millions to billions of neurons.  Those dead neurons are likely the reason for the increased mortality.  Do you not understand cause and effect?

Efficacy and safety of very early rehabilitation for acute ischemic stroke: a systematic review and meta-analysis

Provisionally accepted

Ying Lou Zhongshuo Liu Yingxiao Ji Jinming Cheng Congying Zhao Litao Li ^*

• Hebei General Hospital, Shijiazhuang, China

The final, formatted version of the article will be published soon.

Background: 

Early rehabilitation after acute ischemic stroke (AIS) contributes to functional recovery. However, the optimal time for starting rehabilitation remains a topic of ongoing investigation. This article aims to shed light on the safety and efficacy of very early rehabilitation (VER) initiated within 48 hours of stroke onset. 

Methods: 

A systematic search in PubMed, Embase, Cochrane Library, and Web of Science databases was conducted from inception to January 20, 2024. Relevant literature on VER in patients with AIS was reviewed and the data related to favorable and adverse clinical outcomes were collected for meta-analysis. Subgroup analysis was conducted at different time points, namely at discharge and at three and 12 months. Statistical analyses were performed with the help of the Meta Package in STATA Version 15.0. 

Results: 

A total of 14 randomized controlled trial (RCT) studies and 3,039 participants were included in the analysis. VER demonstrated a significant association with mortality [risk ratio (RR) = 1.27, 95% confidence interval (CI) (1.00, 1.61)], ability of daily living [weighted mean difference (WMD) = 6.90, 95% CI (0.22, 13.57)], and limb motor function [WMD = 5.02, 95% CI (1.63, 8.40)]. However, no significant difference was observed between the VER group and the control group in adverse events [RR = 0.89, 95% CI (0.79, 1.01)], severity of stroke [WMD = 0.52, 95% CI (-0.04, 1.08)], degree of disability [RR = 1.06, 95% CI (0.93, 1.20)], or recovery of walking [RR = 0.98, 95% CI (0.94, 1.03)] after stroke. Subgroup analysis revealed that VER reduced the risk of adverse events in the late stage (at three and 12 months) [RR = 0.86, 95% CI (0.74, 0.99)] and degree of disability at 12 months [RR = 1.28, 95% CI (1.03, 1.60)], and improved daily living ability at three months [WMD=4.26, 95% CI (0.17,8.35)], while increasing severity of stroke during hospitalization [WMD=0.81, 95% CI (0.01, 1.61)]. Conclusion: VER improves activities of daily living (ADLs) and lowers the incidence of long-term complications in stroke survivors. However, premature or overly intense rehabilitation may increase mortality in patients with AIS during the acute phase. PROSPERO registration number: CRD42024508180.

Keywords: Rehabilitation, Early Ambulation, ischemic stroke, prognosis, Meta-analysis

Received: 03 May 2024; Accepted: 09 Oct 2024.

Copyright: © 2024 Lou, Liu, Ji, Cheng, Zhao and Li. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

* Correspondence: Litao Li, Hebei General Hospital, Shijiazhuang, China 

Daily Naps and Brain Training Reduce Dementia Risk

Didn't seem to be allowed while in the hospital. With my vast fatigue I would sleep for the 10 minutes between therapy sessions.

This research has the following notes; 

Daytime naps may be good for our brains, study says 

On average, the difference in brain volume between nappers and non-nappers was equivalent to 2.5 to 6.5 years of aging, researchers said.(So enough to recover the  lost 5 cognitive years from your stroke)

“Our findings suggest that, for some people, short daytime naps may be a part of the puzzle that could help preserve the health of the brain as we get older,” said senior author Victoria Garfield, a senior research fellow at UCL, in a statement. 

And then there is this fucking stupidity:

Frequent daytime naps potential causal risk factor for hypertension, ischemic stroke

 

 And you're that stupid that you think napping is the problem? Rather than figuring the real cause of what is causing the napping.  I'd have you all fired for incompetence, not understanding cause and effect. My god, the stupidity out there is excessive.

And more stupidity:

So set your timer to 59 minutes, although I don't think napping causes Alzheimers.

Daily napping among older adults linked to a 40% higher risk for Alzheimer’s disease

 

The latest here:

Daily Naps and Brain Training Reduce Dementia Risk

Summary: Exercising our brains with daily habits like naps and memory workouts, instead of relying on smartphones, can reduce the risk of age-related dementia. A new study highlights the superiority of human intelligence over AI.

The study emphasizes nurturing our brain’s potential for healthy aging. It also offers practical tips for boosting brain power and maintaining real intelligence.

Key Facts:

1. Daily brain exercises and naps can reduce the risk of dementia.
2. Elmasry’s book emphasizes the unmatched capabilities of human intelligence.
3. Practical tips include associative memory-building and healthy lifestyle choices.

Source: Taylor and Francis Group

People can reduce their risk of age-related dementia by exercising their brains properly instead of Googling, according to a leading Canadian academic.

Professor Mohamed I. Elmasry says simple daily habits such as afternoon naps, memory ‘workouts’ and not reaching for a smartphone can increase the odds of healthy aging.

His new book, iMind: Artificial and Real Intelligence (with foreword by Canadian cell biologist Dr. Aileen Burford-Mason), says the focus has shifted too far away from RI (natural, or real) intelligence in favor of AI (machine, or artificial) intelligence. Elmasry instead calls us to nurture our human mind which, like smartphones, has ‘hardware’, ‘software’ and ‘apps’ but is many times more powerful – and will last much longer with the right care.

As well as napping to refresh our memories and other brain and body functions, he also outlines a series of practical tips to boost brain power and enhance our RI (Real Intelligence). Credit: Neuroscience News

Professor Elmasry, an internationally recognized expert in microchip design and AI, was inspired to write the book after the death of his brother-in-law from Alzheimer’s and others very close to him, including his mother, from other forms of dementia.

Although he says that smart devices are ‘getting smarter all the time’, he argues in iMind that none comes close to ‘duplicating the capacity, storage, longevity, energy efficiency, or self-healing capabilities of the original human brain-mind’.

He writes that: “The useful life expectancy for current smartphones is around 10 years, while a healthy brain-mind inside a healthy human body can live for 100 years or longer.

“Your brain-mind is the highest-value asset you have, or will ever have. Increase its potential and longevity by caring for it early in life, keeping it and your body healthy so it can continue to develop.

“Humans can intentionally develop and test their memories by playing ‘brain games,’ or performing daily brain exercises. You can’t exercise your smartphone’s memory to make it last longer or encourage it to perform at a higher level.”

In iMind: Artificial and Real Intelligence Professor Elmasry shares an anecdote about his grandchildren having to use the search engine on their smartphones to name Cuba’s capital—they had just spent a week in the country with their parents.

The story illustrates how young people have come to rely on AI smartphone apps instead of using their real intelligence (RI), he says, adding: “A healthy memory goes hand-in-hand with real intelligence. Our memory simply can’t reach its full potential without RI.”

Published by Routledge, iMind: Artificial and Real Intelligence includes extensive background on the history of microchip design, machine learning and AI and their role in smartphones and other technology.

The book also explains how both AI and human intelligence really work, and how brain function links the mind and memory. It compares the human mind and brain function with that of smartphones, ChatGPT and other AI-based systems.

Drawing on comprehensive existing research, iMind aims to narrow the knowledge gap between real and artificial intelligence, to address the current controversy around AI, and to inspire researchers to find new treatments for Alzheimer’s, other neurodegenerative conditions and cancer.

It argues that current or even planned AI cannot match the capabilities of the human brain-mind for speed, accuracy, storage capacity and other functions. Healthy aging, Professor Elmasry notes, is as important as climate change but doesn’t attract a fraction of the publicity.

He calls for policymakers to adopt a series of key reforms to promote healthy aging. Among such changes, he suggests that bingo halls could transition from their sedentary entertainment function to become active and stimulating learning centers.

As well as napping to refresh our memories and other brain and body functions, he also outlines a series of practical tips to boost brain power and enhance our RI (Real Intelligence).

These include building up ‘associative’ memory – the brain’s ‘dictionary of meaning’ where it attaches new information to what it already knows. Try reading a book aloud, using all of your senses instead of going on autopilot and turning daily encounters into fully-lived experiences.

Other techniques include integrating a day for true rest into the week, reviewing your lifestyle as early as your 20s or 30s, adopting a healthy diet, and eliminating or radically moderating alcohol consumption to reduce the risk of dementia.

About this dementia and aging research news

Author: Becky Parker-Ellis
Source: Taylor and Francis Group
Contact: Becky Parker-Ellis – Taylor and Francis Group
Image: The image is credited to Neuroscience News

Original Research: “iMind: Artificial and Real Intelligence 1st Edition” by Mohamed I. Elmasry is available for purchase online.