Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Showing posts with label hip fractures. Show all posts
Showing posts with label hip fractures. Show all posts

Tuesday, February 6, 2024

The Effect of Fall Biomechanics on Risk for Hip Fracture in Older Adults: A Cohort Study of Video-Captured Falls in Long-Term Care

What are the EXACT FALL PREVENTION AND PERTURBATION PROTOCOLS from your doctor and therapists? Don't have any? You don't have a functioning stroke doctor or hospital!

The Effect of Fall Biomechanics on Risk for Hip Fracture in Older Adults: A Cohort Study of Video-Captured Falls in Long-Term Care

First published: 13 May 2020
Citations: 41

ABSTRACT

Over 95% of hip fractures in older adults are caused by falls, yet only 1% to 2% of falls result in hip fracture. Our current understanding of the types of falls that lead to hip fracture is based on reports by the faller or witness. We analyzed videos of real-life falls in long-term care to provide objective evidence on the factors that separate falls that result in hip fracture from falls that do not. Between 2007 and 2018, we video-captured 2377 falls by 646 residents in two long-term care facilities. Hip fracture was documented in 30 falls. We analyzed each video with a structured questionnaire, and used generalized estimating equations (GEEs) to determine relative risk ratios (RRs) for hip fracture associated with various fall characteristics. All hip fractures involved falls from standing height, and pelvis impact with the ground. After excluding falls from lower than standing height, risk for hip fracture was higher for sideways landing configurations (RR = 5.50; 95% CI, 2.36–12.78) than forward or backward, and for falls causing hip impact (3.38; 95% CI, 1.49–7.67). However, hip fracture risk was just as high in falls initially directed sideways as forward (1.14; 95% CI, 0.49–2.67), due to the tendency for rotation during descent. Falling while using a mobility aid was associated with lower fracture risk (0.30; 95% CI, 0.09–1.00). Seventy percent of hip fractures involved impact to the posterolateral aspect of the pelvis. Hip protectors were worn in 73% of falls, and hip fracture risk was lower in falls where hip protectors were worn (0.45; 95% CI, 0.21–0.99). Age and sex were not associated with fracture risk. There was no evidence of spontaneous fractures. In this first study of video-captured falls causing hip fracture, we show that the biomechanics of falls involving hip fracture were different than nonfracture falls for fall height, fall direction, impact locations, and use of hip protectors. © 2020 The Authors. Journal of Bone and Mineral Research published by American Society for Bone and Mineral Research.

Introduction

Falls cause 95% of hip fractures in older adults.1, 2 Nearly 25% of hip fracture patients will die within 1 year of the fracture, and 50% will have major declines in independence.3, 4 The rates of falls and hip fractures are especially high among older adults in long-term care (LTC). Only 6% of older adults in Canada live in LTC, but this population experiences 30% of hip fractures.5, 6 Strategies that are effective in preventing falls in community-dwelling seniors (eg, exercise) have been unsuccessful in LTC, due to the high prevalence of physical and cognitive impairment in residents.7 This highlights the importance of complementary strategies in LTC for preventing hip fracture in the event of a fall, through approaches such as vitamin D and calcium supplementation, pharmacologic therapy, and the use of wearable hip protectors,5, 8 which may be especially beneficial for individuals with low body mass index (BMI) who have higher risk for fracture.9-11 Improved understanding of the factors that separate injurious and noninjurious falls may lead to refinements to existing strategies, and new approaches to prevention.

Previous case-control studies in older adults have found that risk for hip fracture in a fall depends at least as much on the biomechanics of the fall, as it does on bone density.12, 39 In particular, although a one standard deviation (1SD) decline in bone density increased fracture risk twofold to threefold,12 falling sideways increased fracture risk sixfold,13 and landing on the hip increased fracture risk 30-fold.14 Furthermore, hip fractures were less common when the person landed on their hand, or grabbed or hit an object to break the fall.39 However, a major limitation of these studies is their reliance on interviews or questionnaires completed by the faller and witnesses (if any) to determine a narrow range of fall characteristics (fall direction, fall height, and contact sites). Recalling the circumstances of falls is challenging, especially for older adults with cognitive impairment.15, 16 Also, fallers who sustained a fracture may bias their response based on the notion that they must have landed on the hip for fracture to occur.11 To date, objective evidence on the circumstances of falls causing hip fracture has not been available to overcome these limitations.

In this study, we address this knowledge gap by analyzing videos of real-life falls experienced by older adults living in LTC, and comparing the characteristics of falls that did result in hip fracture versus those that did not result in hip fracture. We hypothesized that fracture risk would associate with fall characteristics that have previously been shown to be important, as reviewed above (fall height, fall direction, contact sites, use of hip protectors, and BMI). We considered initial fall direction and landing configuration separately, based on our previous observation that falls in LTC often involve rotation during descent.17 In addition to hip and hand impact, we examined knee impact (which may decrease impact severity to the hip), use of mobility aids, and attempts to recover balance by stepping, which we previously found to decrease hip impact velocity.18 We also tested whether fracture risk, as hypothesized by Cummings and Nevitt,19 associated with activity at the time of the fall (eg, walking versus standing), and with cause of imbalance.

 
More at link.

Sunday, March 15, 2020

Long-Term Risk of Hip Fracture After Ischemic Stroke

My hip must be pretty strong because this incident did not result in a broken hip.  I've fallen on it many times since then and making it stronger each time it occurs.

Epic failure at bike stroke therapy

Similar to boxers microfracturing their hands so they grow back stronger. I should be able to ask my doctor specifically if the hip is now strong enough to survive a fall. Don't listen to me, I'm not medically trained.

 

Long-Term Risk of Hip Fracture After Ischemic Stroke 

First Published June 30, 2019 Research Article




Data are lacking on the long-term risk of hip fracture among patients with ischemic stroke. A better understanding of the contemporary incidence of hip fracture after ischemic stroke could benefit patient care by promoting strategies to prevent this disabling complication in stroke survivors.

We performed a retrospective cohort study using inpatient and outpatient claims between 2008 and 2015 from a nationally representative 5% sample of Medicare beneficiaries. We included patients ≥66 years of age who were hospitalized with acute ischemic stroke, defined through a validated diagnostic code algorithm. We excluded patients who had a prior or concurrent hip fracture diagnosis at the time of ischemic stroke. The primary outcome was hip fracture requiring hospitalization. Survival statistics were used to calculate crude incidence rates, and the Kaplan-Meier method was used to calculate cumulative rates.

Among the 1 772 550 beneficiaries in our sample, 60 099 were diagnosed with an acute ischemic stroke without prior or concomitant hip fracture. During 4.5 (±2.2) years of follow-up, the incidence of hip fracture was 1.6 (95% confidence interval [CI]: 1.5-1.6) per 100 person-years in patients with acute ischemic stroke versus 0.6 (95% CI: 0.6-0.6) per 100 person-years in patients without acute ischemic stroke. The cumulative 5-year rate of hip fracture was 7.6% (95% CI: 7.2%-8.0%) among patients with acute ischemic stroke versus 2.8% (95% CI: 2.8%-2.9%) among the remaining Medicare beneficiaries.

We found that among elderly Medicare beneficiaries with acute ischemic stroke, nearly 1 in 12 developed a hip fracture over the next 5 years.

Wednesday, July 3, 2019

Long-Term Risk of Hip Fracture After Ischemic Stroke

I went down the route of falling on my affected hip numerous times in order to strengthen it. Similar to boxers microfracturing their hands so they grow back stronger. I should be able to ask my doctor specifically if the hip is now strong enough to survive a fall. Don't listen to me, I'm not medically trained. Is your doctor able to answer any of your stroke questions with factual answers?  A better use of these research dollars would have been to come up with fall prevention protocols. 

Long-Term Risk of Hip Fracture After Ischemic Stroke 

First Published June 30, 2019 Research Article




Data are lacking on the long-term risk of hip fracture among patients with ischemic stroke. A better understanding of the contemporary incidence of hip fracture after ischemic stroke could benefit patient care by promoting strategies to prevent this disabling complication in stroke survivors.

We performed a retrospective cohort study using inpatient and outpatient claims between 2008 and 2015 from a nationally representative 5% sample of Medicare beneficiaries. We included patients ≥66 years of age who were hospitalized with acute ischemic stroke, defined through a validated diagnostic code algorithm. We excluded patients who had a prior or concurrent hip fracture diagnosis at the time of ischemic stroke. The primary outcome was hip fracture requiring hospitalization. Survival statistics were used to calculate crude incidence rates, and the Kaplan-Meier method was used to calculate cumulative rates.

Among the 1 772 550 beneficiaries in our sample, 60 099 were diagnosed with an acute ischemic stroke without prior or concomitant hip fracture. During 4.5 (±2.2) years of follow-up, the incidence of hip fracture was 1.6 (95% confidence interval [CI]: 1.5-1.6) per 100 person-years in patients with acute ischemic stroke versus 0.6 (95% CI: 0.6-0.6) per 100 person-years in patients without acute ischemic stroke. The cumulative 5-year rate of hip fracture was 7.6% (95% CI: 7.2%-8.0%) among patients with acute ischemic stroke versus 2.8% (95% CI: 2.8%-2.9%) among the remaining Medicare beneficiaries.

We found that among elderly Medicare beneficiaries with acute ischemic stroke, nearly 1 in 12 developed a hip fracture over the next 5 years.

Thursday, August 7, 2014

Hip fractures after stroke and their prevention

It has only been 7 years since this came out so I'm sure your doctor has created a stroke protocol to address this problem. You can stop laughing now.
http://qjmed.oxfordjournals.org/content/100/9/539.short
  1. QJM 100 (9): 539-545. doi: 10.1093/qjmed/hcm067

Abstract

Increased fracture risk is a recognized complication following stroke. Bone loss following a hemiplegic stroke has been proposed as a major risk factor for post-stroke hip fracture, with a recent focus on the development of novel therapeutic measures to prevent bone loss and fractures after stroke. We briefly review the literature on the epidemiology and pathophysiology of bone loss and hip fracture after stroke, and then critically review recent studies on preventive strategies. 


Sunday, July 28, 2013

Effect of Folate and Mecobalamin on Hip Fractures in Patients With Stroke

Only 8 years old. How incompetent is your stroke hospital if they haven't put this into place yet? Seems like a fireable offense for the hospital board of directors to enforce. Why would you even go to a hospital if they can't even read and apply research  to their areas of expertise? The following of Joint Commission standards is even more of a reason to never set foot in such a hospital. No innovation. But then I'm a stupid non-medical stroke survivor, they would have to be stupider than me to not see  where they can apply research to stroke protocols.
http://jama.jamanetwork.com/article.aspx?articleid=200453
Context  Stroke increases the risk of subsequent hip fracture by 2 to 4 times. Hyperhomocysteinemia is a risk factor for both ischemic stroke and osteoporotic fractures in elderly men and women. Treatment with folate and mecobalamin (vitamin B12) may improve hyperhomocysteinemia.
Objective  To investigate whether treatment with folate and vitamin B12 reduces the incidence of hip fractures in patients with hemiplegia following stroke.
Design, Setting, and Patients  A double-blind, randomized controlled study of 628 consecutive patients aged 65 years or older with residual hemiplegia at least 1 year following first ischemic stroke, who were recruited from a single Japanese hospital from April 1, 2000, to May 31, 2001. Patients were assigned to daily oral treatment with 5 mg of folate and 1500 μg of mecobalamin, or double placebo; 559 completed the 2-year follow-up.
Main Outcome Measure  Incidence of hip fractures in the 2 patient groups during the 2-year follow-up.
Results  At baseline, patients in both groups had high levels of plasma homocysteine and low levels of serum cobalamin and serum folate. After 2 years, plasma homocysteine levels decreased by 38% in the treatment group and increased by 31% in the placebo group (P<.001). The number of hip fractures per 1000 patient-years was 10 and 43 for the treatment and placebo groups, respectively (P<.001). The adjusted relative risk, absolute risk reduction, and the number needed to treat for hip fractures in the treatment vs placebo groups were 0.20 (95% confidence interval [CI], 0.08-0.50), 7.1% (95% CI, 3.6%-10.8%), and 14 (95% CI, 9-28), respectively. No significant adverse effects were reported.
Conclusion  In this Japanese population with a high baseline fracture risk, combined treatment with folate and vitamin B12 is safe and effective in reducing the risk of a hip fracture in elderly patients following stroke.
The risk of a hip fracture in patients after stroke is 2 to 4 times higher than that in age-matched healthy control patients.1 These fractures usually occur relatively late after stroke onset and affect the paretic side of the body.2- 5 Hip fractures are associated with more deaths, disabilities, and medical costs than all other osteoporosis-related fractures combined.6 We previously measured the bone mineral density (BMD) in patients with stroke in the second metacarpal bone and demonstrated a decrease in the bone mass in the hemiplegic limb that corresponded to the degree of palsy and vitamin D deficiency,7 which may explain why hip fractures in patients poststroke occur almost exclusively on the hemiplegic side of the body.
A close association between plasma homocysteine and risk of ischemic stroke has been reported,8- 11 and plasma homocysteine levels are higher in patients with ischemic stroke in both acute12- 13 and convalescent phases.14- 17 In patients with homocysteinuria, a rare autosomal recessive biochemical abnormality, there is an increased prevalence of skeletal abnormalities,18- 20 including osteoporosis, a primary risk factor for hip fracture. Thus, elevated plasma homocysteine concentrations may be associated with osteoporosis and increase the risk of a hip fracture. An increased homocysteine level appears to be a strong and independent risk factor for an osteoporotic fracture of the bones, including the hip, in older men and women.21- 22
In the remethylation cycle, homocysteine is salvaged for methionine synthesis by the addition of a methyl group by methionine synthase.23 Vitamin B12 (cobalamin) is an essential cofactor for methionine synthase and N5-methyl-tetrahydrofolate serves as the methyl donor. Therefore, there are close relationships between plasma homocysteine and cobalamin and folate.8- 9,24- 26
We previously demonstrated a reduction in plasma homocysteine levels by combination therapy with folate and vitamin B12 in patients with ischemic stroke.26 Our goal for this study was to investigate the efficacy of the combined therapy for decreasing the risk of fractures, particularly in the hip, in a 2-year trial in elderly patients with hemiplegia following ischemic stroke.