Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, July 17, 2024

The Role of Coenzyme Q10 in Cardiovascular Disease Treatment: An Updated 2024 Systematic Review and Meta-Analysis of Prospective Cohort Studies (2000-2023)

What does your doctor have to say about this?
Can coenzyme Q10 reduce the risk of side effects from statins?

The latest here:

 The Role of Coenzyme Q10 in Cardiovascular Disease Treatment: An Updated 2024 Systematic Review and Meta-Analysis of Prospective Cohort Studies (2000-2023)

Julian Yin Vieira Borges

Abstract 

Background: 
 
Cardiovascular diseases (CVDs) are the leading cause of death globally. Mitochondrial dysfunction is implicated in the pathogenesis of various CVDs. Coenzyme Q10 (CoQ10), a crucial component of the mitochondrial electron transport chain, has potential cardioprotective effects. This study aims to evaluate the efficacy of adding CoQ10 nutrient supplementation in the Treatment of Cardiovascular Disease Protocols in patients with CVDs. 
 
Methods: 
 
This systematic review and meta-analysis assessed data from PubMed, Scopus, and Cochrane Library databases up to January 2024. Prespecified inclusion criteria were randomized controlled trials (RCTs) involving adult patients with CVDs, comparing CoQ10 supplementation with placebo or standard care. Exclusion criteria included non-randomized studies, studies without a control group, and those not reporting relevant outcomes. Data extraction and quality assessment were performed independently by two reviewers. The primary outcome was improvement in mitochondrial function, assessed through ATP production and mitochondrial respiratory capacity. Secondary outcomes included changes in systolic function, measured by ejection fraction. Statistical analysis was conducted using Review Manager software. 
 
Findings: 
 
The search identified 243 studies, of which 5 RCTs met the inclusion criteria, encompassing a total of 1,200 participants (600 male, 600 female). CoQ10 supplementation significantly improved mitochondrial function in cardiomyocytes, evidenced by increased ATP production and enhanced mitochondrial respiratory capacity. Additionally, CoQ10 was associated with a significant improvement in systolic function, measured by ejection fraction (mean difference: 5.6%, 95% CI: 3.2% to 8.0%, p<0.001). The risk of bias across studies was generally low, with an I statistic of 25% indicating low heterogeneity. Interpretation: This meta-analysis supports the beneficial effects of CoQ10 on mitochondrial function and systolic performance in patients with CVD. These findings suggest that CoQ10 supplementation could be a valuable adjunctive therapy in this population. However, further large-scale RCTs are necessary to confirm these results and determine the optimal dosage and duration of therapy. 
 
Keywords: Coenzyme Q10, Ubiquinone, Cardiovascular diseases, Heart failure, Myocardial infarction,
Coronary artery disease, Mitochondrial dysfunction, Ejection fraction, Endothelial function
Cardiovascular Disease
Cardiology

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