Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Tuesday, January 29, 2019

Increased intrinsic connectivity for structural atrophy and functional maintenance after acute ischemic stroke

I got nothing out of this.  How the hell do you increase intrinsic connectivity? The result of this research is that 10 million yearly stroke survivors  will need to set up their own research to figure this out. That is the height of insanity.   The stroke medical world seems to have fewer brains than those 10 million yearly stroke survivors.

Increased intrinsic connectivity for structural atrophy and functional maintenance after acute ischemic stroke


First published: 25 January 2019
This article has been accepted for publication and undergone full peer review but has not been through the copyediting, typesetting, pagination and proofreading process, which may lead to differences between this version and the Version of Record. Please cite this article as doi: 10.1111/ene.13913



Abstract

Background

Significant clinical recovery has been commonly observed in ischemic stroke patients with irreversible brain structural damage. However, brain mechanisms that help to maintain clinical function remain unclear.

Methods

Sixty‐two patients with acute ischemic stroke underwent longitudinal clinical assessments and MRI scanning. We evaluated clinical recovery trajectory using Hierarchical Linear Model and analysed intrinsic connectivity with a seed‐based approach to examine its changing pattern based on the regional volume changes calculated using voxel‐wise analysis.

Results

We observed that clinical outcome measures improved mainly in the short‐term period (baseline vs. 3 weeks) and then remained stable. Gray matter volume was reduced significantly in bilateral caudate over the entire 3‐year long‐term period. Significant intrinsic connectivity increases in caudate‐middle cingulum over the short‐term period and in caudate‐precuneus and caudate‐calcarine over the long‐term period were observed. Finally, we found that increased caudate‐calcarine connectivity was associated with reduced right caudate volume, and a positive correlation was found between increased caudate‐middle cingulum connectivity and the amount of the modified Rankin score changes.

Conclusions

Increased intrinsic connectivity found in this study tends to be a compensatory mechanism for post‐stroke structural damage, associated with clinical recovery. This study helped in understanding the significance(But did nothing to help survivors) of enhanced intrinsic connectivity in post‐stroke long‐term assessment and rehabilitation.
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