Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Sunday, March 20, 2022

A simple score to predict atherosclerotic or embolic intracranial large-vessel occlusion stroke before endovascular treatment

 So you're identifying the cause of the stroke but you didn't take the obvious next step to CREATE EXACT PROTOCOLS THAT PREVENT THE NEXT STROKE.

A simple score to predict atherosclerotic or embolic intracranial large-vessel occlusion stroke before endovascular treatment

Geng Liao MD1, Zhenyu Zhang MBBS1, Tao-Hsin Tung PhD4, Yuemei He MPA2, Linhui Hu MD2, Xiong Zhang MD1, Hai Chen MBBS1, Jinbo Huang MBBS1, Weijie Du MBBS1, Chaomao Li MBBS1, Zhi Yang MBBS1, Yong Cai MBBS3, and Hanxiang Liang MD3
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Online Publication Date:
18 Mar 2022
Page Range:
1–8
Volume/Issue:
Publish Before Print
DOI link:
https://doi.org/10.3171/2022.1.JNS212924
Restricted access

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OBJECTIVE

The authors developed a method to predict the etiology of intracranial large-vessel occlusion stroke (ILVOS) before endovascular treatment.

METHODS

The authors retrospectively evaluated two etiologies of ILVOS—intracranial atherosclerotic stenosis–related occlusion (ICAS-O) and embolism-related occlusion (EMB-O)—in a cohort of patients from the National Comprehensive Stroke Center database of China. Patients were randomly divided into the derivation and validation cohorts at a ratio of 2:1. The authors derived the score in the derivation cohort and assessed the score in the validation cohort.

RESULTS

The authors identified 608 of 662 patients with ILVOS who received endovascular treatment during the study period. After adjustment for confounding factors, hypertension (OR 2.90, 95% CI 1.34–6.26), diabetes mellitus (OR 2.80, 95% CI 1.45–5.42), absence of atrial fibrillation (OR 27.29, 95% CI 13.27–56.09), National Institutes of Health Stroke Scale score < 7 (OR 2.92, 95% CI 1.22–6.99), and absence of the computed tomography hyperdense sign (OR 2.86, 95% CI 1.22–6.74) were significantly related to ICAS-O. A score was derived to help predict ICAS-O or EMB-O. The area under the curve values of the receiver operating characteristic curve for ICAS-O identification were 0.886 (95% CI 0.839–0.933) and 0.880 (95% CI 0.846–0.914) in the derivation and validation cohorts, respectively.

CONCLUSIONS

The atrial fibrillation–blood pressure–clinical neurological deficit–computed tomography hyperdense sign–diabetes mellitus (ABC2D) score can be used to identify atherosclerotic or embolic etiology of patients with ILVOS who require emergency endovascular treatment.

 
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