Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Monday, March 21, 2022

MI tied to reduced risk for Parkinson’s disease

This is the opposite of stroke survivors, so you'll need EXACT PARKINSON PREVENTION PROTOCOLS post stroke. 

Your risk of Parkinsons here:

Parkinson’s Disease May Have Link to Stroke March 2017 

I guess you could do coffee but you'll have to ask your doctor for specific amounts and decaf or regular.  But don't listen to me, I'm not medically trained.

How coffee protects against Parkinson’s Aug. 2014 

The latest here:

MI tied to reduced risk for Parkinson’s disease

One-year survivors of MI experienced decreased risk for developing Parkinson’s disease and secondary Parkinsonism compared with the general population, researchers reported.

According to a study published in the Journal of the American Heart Association, the association between MI and reduced risk for Parkinson’s disease may be indicative of an inverse relationship between Parkinson’s disease and CV risk factors.

Parkinson's disease
Source: Adobe Stock

“Parkinson’s disease is primarily a neurodegenerative disease, whereas Parkinsonism has several underlying causes besides primary neurodegenerative processes, including a variety of vascular mechanisms. These may include lacunar infarcts and other vascular insults encompassing the substantia nigra, cerebral white matter and other cerebral structures,” Jens Sundbøll, MD, PhD, from the departments of clinical epidemiology and cardiology at the Aarhus University Hospital, Denmark, and colleagues wrote. “We therefore examined the long-term risk of Parkinson’s disease and secondary Parkinsonism following first-time MI and the impact of common MI treatments and complications.”

Utilizing Danish medical registries, researchers compared the incidence of Parkinson’s disease and secondary Parkinsonism at 1 year among patients with a first-time MI diagnosis from 1995 to 2016 and a sex- , age- and calendar year-matched general population cohort without MI.

MI and subsequent Parkinson disease risk

A total of 181,994 patients were included in the MI cohort (median age at MI diagnosis, 71 years; 62% men) and 909,970 matches were included from the general population.

After 21 years of follow-up, researchers reported that the cumulative incidence of Parkinson’s disease was 0.9% in the MI cohort and the incidence of secondary Parkinsonism was 0.1% compared with 1.25% and 0.14%, respectively, in the general population.

According to the study, occurrence of MI was associated with lower risk for both Parkinson’s disease (adjusted HR = 0.8; 95% CI, 0.73-0.87) and secondary Parkinsonism (aHR = 0.72; 95% CI, 0.54-0.94).

MI subgroup analyses

Sundbøll and colleagues observed no impact of procedures and complications after MI on subsequent risk for Parkinson disease or secondary Parkinsonism and no difference when patients and the general population were stratified by sex.

Researchers noted that the reduced risk for Parkinson’s disease was more pronounced among older patients.

According to the study, these findings indicate that an unmeasured confounder would need to be strongly associated with both MI and Parkinson’s disease to explain these results. Potential remains for a confounder with a moderate to large effect, such as smoking, to explain the results of the present analysis.

“There are very few diseases in this world in which smoking decreases risk: Parkinson’s disease is one, and ulcerative colitis is another. Smoking increases the risk of the most common diseases including cancer, cardiovascular disease and pulmonary disease and is definitely not good for your health,” Sundbøll said in a press release.

Researchers reported no effect of cardiac and noncardiac comorbidity, Charlson Comorbidity Index or use of drugs associated with extrapyramidal adverse effects mimicking Parkinson’s disease on the observed association.

Moreover, the type of MI experienced by individuals in the MI cohort did not substantially impact the results.

“For physicians treating patients following a heart attack, these results indicate that cardiac rehabilitation should be focused on preventing ischemic stroke, vascular dementia and other cardiovascular diseases such as a new heart attack and heart failure, since the risk of Parkinson’s appears to be decreased in these patients, in comparison to the general population,” Sundbøll said in the release.

Reference:

 

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