Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Tuesday, April 19, 2022

Teaching NeuroImages: Crying thalamus Manifestation of acute ischemic stroke

 So you described a problem, offered no solution. Useless

Teaching NeuroImages: Crying thalamus Manifestation of acute ischemic stroke

Ahmed Z. Obeidat, Joseph P. Broderick

A 58-year-old right-handed man with hyperlipidemia presented with 2 brief episodes of explosive crying and confusion. Physical examination was unremarkable. MRI showed patchy acute ischemic stroke within the left anterior thalamus and magnetic resonance angiography showed stenosis of the P2 segment of the left posterior cerebral artery (figure), likely affecting the thalamogeniculate perforator. Pathologic crying has been termed folles larmes prodromiques and has been described in association with various CNS lesions including thalamic.1,2 Acute pathologic crying remains a rare presentation of stroke and may easily be overlooked. This report serves to remind clinicians of the importance of detailed neurologic assessment under such circumstances.

Figure Acute thalamic stroke and localized vascular stenosis

Brain MRI shows (A) diffusion-weighted imaging with diffusion restriction consistent with patchy acute ischemic stroke within the anterior thalamus (right brace) and (B) accompanying apparent diffusion coefficient signal reduction (right brace). (C, D) Magnetic resonance angiography head without contrast shows flow signal drop consistent with stenosis of the P2 segment of the left posterior cerebral artery (red arrows).

 

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