Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Monday, July 12, 2021

ASCOT: Amlodipine outperforms atenolol in stroke prevention

 I had to get off of Amlodipine since it gave me edema in my left side affected ankle. Took weeks after being off before it returned to normal. Now on nifedipine.

ASCOT: Amlodipine outperforms atenolol in stroke prevention

Long-term results from the ASCOT trial demonstrated that an amlodipine-based BP-lowering regimen reduced stroke risk compared with an atenolol-based regimen, although no benefit in dementia risk was observed.

Since management of stroke risk factors may reduce later dementia, William N. Whiteley, PhD, Scottish senior clinical fellow in Centre for Clinical Brain Sciences at the University of Edinburgh, U.K., and colleagues evaluated whether dementia or stroke were linked to different BP-lowering regimens; atorvastatin or placebo; and mean BP, BP variability and mean cholesterol levels.

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In the ASCOT trial, patients with hypertension and at least three CVD risk factors were randomly assigned to an amlodipine- or atenolol-based BP-lowering regimen targeting a BP less than 140/90 mm Hg for 5.5 years. The researchers also randomly assigned patients with total cholesterol of 6.5 mmol/L or less to atorvastatin 10 mg or placebo for 3.3 years.

Of the 8,580 U.K. participants, researchers followed 7,300 for up to 21 years from randomization, with a median follow-up of 17 years. The mean age was 64 years, and most patients were men (81% in BP-lowering arm; 87% in lipid-lowering arm).

Results revealed that atorvastatin use for 3.3 years failed to affect stroke (adjusted HR = 0.92; 95% CI, 0.78-1.09; P = .341) or dementia (aHR = 0.98; 95% CI, 0.82-1.18; P = .837) compared with placebo. No associations between mean total cholesterol and later stroke or dementia were reported.

Moreover, compared with an atenolol-based regimen, an amlodipine-based regimen for 5.5 years lowered the risk for stroke (aHR = 0.82; 95% CI, 0.72-0.93; P = .003) but not dementia (aHR = 0.94; 95% CI, 0.82-1.07; P = .334) during follow-up.

In other data, BP variability conferred a higher risk for dementia (HR per 5 mm Hg = 1.14; 95% CI, 1.06-1.24; P < .001) and stroke (per 5 mm Hg, HR = 1.21; 95% CI, 1.12-1.32; P < .001), adjusted for mean BP.

“Higher BP variability,” the researchers wrote, “is largely due to age-related stiffening of large arteries and loss of baroreflex function. Antihypertensive drugs have little beneficial effect in reducing variability, although dihydropyridine calcium channel blockers may have a modest effect.”

In conclusion, they wrote: “We demonstrate the importance of BP control with amlodipine rather than atenolol for stroke prevention and that starting amlodipine about 5 years earlier still has an important detectable effect on stroke incidence over 20 years. Despite this reduction in stroke in incidence, there was no reduction in dementia incidence, although dementia was almost as frequent as stroke over follow-up.”

 

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