Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Friday, April 15, 2022

Blood Biomarker Linked With Parkinson's Disease

 You better hope like hell your doctor has a protocol on testing for this post stroke and then has the protocols to prevent Parkinsons. At least if you have a competent doctor and hospital, this will exist.

Your risk of Parkinsons here:

Parkinson’s Disease May Have Link to Stroke March 2017 

The latest here:

Blood Biomarker Linked With Parkinson's Disease

 

Is neurofilament light the C-reactive protein of neurology?

A computer rendering of nerve cells affected by Parkinson’s disease.

Serum neurofilament light (NfL), a measure of axonal injury, was associated with incident Parkinson's disease, parkinsonian signs, and decline in physical functioning, a longitudinal study showed.

Among 1,300 people in the Chicago Health and Aging Project (CHAP), a twofold higher concentration of serum NfL was associated with incident clinical Parkinson's disease (OR 2.54, 95% CI 1.70-3.81), according to Shannon Halloway, PhD, RN, of Rush University Medical Center in Chicago, and co-authors.

Compared with people in the lowest quartile of serum NfL at baseline, those with higher concentrations had greater odds of clinical Parkinson's disease at all time points, ranging from the date of diagnosis to more than 5 years earlier, the researchers reported in Neurology.

"This study confirms the potential of NfL to detect a neurological disease years before the clinical picture becomes manifest, even in the case of Parkinson's disease where cerebrospinal fluid (CSF) and blood NfL do not clearly peak in the clinical phase as in other central nervous system (CNS) degenerative diseases," wrote Lorenzo Gaetani, MD, PhD, and Lucilla Parnetti, MD, PhD, both of the University of Perugia in Italy, in an accompanying editorial.

In recent years, ultrasensitive immunoassays have made it possible to measure NfL in serum and plasma, they observed. "This sentinel of neuronal damage has been shown to detect the presence of a variety of CNS diseases, from multiple sclerosis to Alzheimer's disease, frontotemporal dementia, motor neuron diseases, parkinsonisms, cerebrovascular disease, and traumatic brain injury," they noted.

"Due to its ability to reflect the dynamic nature of CNS injuries, NfL has been called the C-reactive protein of neurology," Gaetani and Parnetti wrote. In a hypothetical future scenario, serum NfL might be a first test, which would be followed by more disease-specific blood or CSF analyses, they added.

Halloway and co-authors studied 1,327 people over age 65 who were enrolled in six triennial cycles of the CHAP cohort study from 1993 to 2012. Blood samples were collected from 1994 to 2012, at both study entry and over time.

Clinical evaluations included assessing parkinsonian signs in four domains -- bradykinesia, parkinsonian gait, rigidity, and tremors -- using a modified version of the Unified Parkinson's Disease Rating Scale (UPDRS). Performance tests evaluated physical functioning, which were scored 0-15, with lower scores indicating potential inability to conduct tasks of independent living. Board-certified neurologists diagnosed Parkinson's disease.

At baseline, participants had an average age of about 74; 62% were women, and 61% were Black. Baseline serum NfL ranged from 0.99 to 755 pg/mL. The highest quartile had NfL values greater than 37.3 pg/mL; in the lowest quartile, NfL was less than 18.5 pg/mL.

During the study, 77 people (6.1%) were diagnosed with clinical Parkinson's disease. Parkinsonian signs were 9.5 on average (range 0-66).

People with NfL values in the highest quartile at baseline had a significantly higher risk of subsequently developing Parkinson's disease compared with those with NfL in the lowest quartile (OR 3.74, 95% CI 1.75-8.0).

Among people with twofold higher concentrations of serum NfL at baseline, the annual rate of physical functioning decline increased by 0.15 units.

People with twofold higher NfL concentrations were also more likely to have parkinsonian signs (OR 2.44, 95% CI 1.94-2.94). There was a significant graded association with parkinsonian signs in all domains in the third (25.4 to 37.3 pg/mL) and fourth NfL quartile.

Findings from the analysis may be limited, the researchers acknowledged. The sample represented a biracial cohort in metropolitan Chicago, and results may not apply to other groups. The neuropathological development of Parkinson's disease may begin in earlier periods not captured in this study. In addition, the recruitment timeline of the study spanned 16 years, which may have resulted in variability of Parkinson's diagnoses over time.

  • Judy George covers neurology and neuroscience news for MedPage Today, writing about brain aging, Alzheimer’s, dementia, MS, rare diseases, epilepsy, autism, headache, stroke, Parkinson’s, ALS, concussion, CTE, sleep, pain, and more. Follow

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