Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Saturday, August 8, 2020

Large-scale informatic analysis to algorithmically identify blood biomarkers of neurological damage

It is incredibly simple, which means our stroke medical professionals will fucking miss it. You take these objective blood testing damage markers and map protocols to them which fix the problems that blood testing has identified. But no one in the stroke medical world has two neurons to rub together and actually help stroke survivors. You're screwed along with your children and grandchildren.  

Large-scale informatic analysis to algorithmically identify blood biomarkers of neurological damage

Grant C. O’Connell, Megan L. Alder, Christine G. Smothers, and Julia H. C. Chang
  1. Edited by Vincent T. Marchesi, Yale University School of Medicine, New Haven, CT, and approved July 9, 2020 (received for review April 23, 2020)

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Significance

The discovery and development of precision blood biomarkers which can accurately detect damage to brain tissue could transform how we diagnose and treat neurological pathologies. In this study, we used mRNA expression data generated from thousands of tissue samples to algorithmically evaluate nearly every protein-coding gene in the human genome in terms of potential to produce blood biomarkers for neurological damage based on expression profiles both across the body and within the brain. This unprecedented analysis identifies a plethora of previously unexplored candidate blood biomarkers which could have clinical utility for noninvasive diagnosis and monitoring of various common neurological conditions, including traumatic brain injury, stroke, and multiple sclerosis.

Abstract

The identification of precision blood biomarkers which can accurately indicate damage to brain tissue could yield molecular diagnostics with the potential to improve how we detect and treat neurological pathologies. However, a majority of candidate blood biomarkers for neurological damage that are studied today are proteins which were arbitrarily proposed several decades before the advent of high-throughput omic techniques, and it is unclear whether they represent the best possible targets relative to the remainder of the human proteome. Here, we leveraged mRNA expression data generated from nearly 12,000 human specimens to algorithmically evaluate over 17,000 protein-coding genes in terms of their potential to produce blood biomarkers for neurological damage based on their expression profiles both across the body and within the brain. The circulating levels of proteins associated with the top-ranked genes were then measured in blood sampled from a diverse cohort of patients diagnosed with a variety of acute and chronic neurological disorders, including ischemic stroke, hemorrhagic stroke, traumatic brain injury, Alzheimer’s disease, and multiple sclerosis, and evaluated for their diagnostic performance. Our analysis identifies several previously unexplored candidate blood biomarkers of neurological damage with possible clinical utility, many of which whose presence in blood is likely linked to specific cell-level pathologic processes. Furthermore, our findings also suggest that many frequently cited previously proposed blood biomarkers exhibit expression profiles which could limit their diagnostic efficacy.

 

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