Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Saturday, September 19, 2020

Risk of Parkinson disease following gout: A population-based retrospective cohort study in Taiwan

But do you want gout because of this?

Gout may lessen Alzheimer risk

The latest here:

Risk of Parkinson disease following gout: A population-based retrospective cohort study in Taiwan

Hu LY, Yang AC, Lee SC, et al
BMC Neurology|September 10, 2020

Researchers used Taiwan’s National Health Insurance Research Database to evaluate the risk of Parkinson disease (PD) in patients suffering from gout. They analyzed data from patients newly diagnosed with gout between January 1, 2000 and December 1, 2000. They performed comparison with a cohort of patients without gout, matched for gender and age. The gout cohort vs the control cohort had no significantly higher hazard ratio for PD, even following adjustment for age, urbanization, monthly income, gender, and comorbidities. There were no gender differences in the gout–PD link. Overall, in the Taiwanese population, it was evident that gout did not confer protection against the risk of PD.

Read the full article on BMC Neurology.

 
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