Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Sunday, October 2, 2016

Flavanone intake is inversely associated with risk of incident ischemic stroke in the REasons for Geographic and Racial Differences in Stroke (REGARDS) study

I think this means more Flavanone intake  results in fewer strokes. Which should mean our fucking failures of stroke associations get cracking on a stroke diet protocol with exact amounts to eat. Research is going to be needed to be followed up using stroke survivors.
http://www.mdlinx.com/internal-medicine/medical-news-article/2016/09/26/ischemic-stroke-flavonoid-plant-based-diet/6871965/?news_id=881&newsdt=100116&subspec_id=488&utm_source=WeeklyNL&utm_medium=newsletter&utm_content=Weeks-Best-Article&utm_campaign=article-section&category=latest-weekly


The Journal of Nutrition, 09/26/2016

In this study, the researchers conclude that greater utilization of flavanones, but not total or other flavonoid subclasses, was contrarily connected with incident ischemic stroke. In ischemic stroke incidence associations did not differ by sex, race, or region for the association; but, regional differences in flavanone intake may contribute to regional disparities. In blacks higher flavanone intake recommends that flavanone intake is not implicated in racial disparities in ischemic stroke incidence.

Methods


  • 20,024 participants were evaluated in the REGARDS (Reasons for Geographic and Racial Differences in Stroke) study, a biracial prospective study.
  • They excluded participants with stroke history(WHY?) or missing dietary data.
  • They estimated flavonoid intake by using a Block98 food frequency questionnaire and the USDA’s Provisional Flavonoid Addendum and Proanthocyanidin Database.
  • They assessed associations between quintiles of flavonoid intake and incident ischemic stroke by using Cox proportional hazards models, adjusting for confounders.

Results


  • 524 acute ischemic strokes happened over 6.5 y.
  • As per the outcomes, flavanone intake was lower in the Southeastern United States but higher in blacks than in whites.
  • Flavanone intake was inversely associated with incident ischemic stroke (HR: 0.72; 95% CI: 0.55, 0.95; P–trend = 0.03), after multivariable adjustment.
  • The study observed that consumption of citrus fruits and juices was inversely associated with incident ischemic stroke (HR: 0.69; 95% CI: 0.53, 0.91; P–trend = 0.02).
  • No association was found between total flavonoids and other flavonoid subclasses with incident ischemic stroke.
  • The study reveal that there was no statistical interaction with sex, race, or region for any flavonoid measure.



Go to PubMed Go to Abstract Print Article Summary Cat 2 CME Report

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