Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, June 4, 2025

Aging Brain More Vulnerable to Even Mild Oxygen Deprivation

 Which makes it even more important that your competent? hospital strive for tPA delivery in 3 minutes!

In this research in mice the needed time frame for tPA delivery is 3 minutes for full recovery. What is your plan to accomplish that? Or are you ignoring that need?

Electrical 'storms' and 'flash floods' drown the brain after a stroke)

The latest here:

Aging Brain More Vulnerable to Even Mild Oxygen Deprivation

Summary: New research shows that even mild, prolonged exposure to low oxygen levels can significantly disrupt the blood-brain barrier (BBB) in aging brains. Using mice aged 2 to 23 months, scientists found that older mice experienced 4–6 times more BBB breakdown than younger mice under similar hypoxic conditions.

The study also revealed increased brain inflammation in older mice, regardless of oxygen level. These findings may explain why elderly individuals with chronic respiratory or cardiovascular issues are at greater risk for neurodegeneration and cognitive decline.

Key Facts:

  • Heightened Vulnerability: Aged mice showed significantly more BBB disruption at higher oxygen levels than younger mice, indicating increased sensitivity.
  • Inflammation Spike: Microglial activation, a marker of brain inflammation, was elevated in older mice even under normal oxygen conditions.
  • Repair Deficit: Despite steady blood vessel growth across ages, older brains exhibited impaired BBB repair, suggesting weakened vascular resilience with age.

Source: Impact Journals

In a new study, researchers Arjun Sapkota, Sebok K. Halder, and Richard Milner from San Diego Biomedical Research Institute investigated how aging affects the brain’s vulnerability to low oxygen, or hypoxia.

Using C57BL/6J mice ranging from 2 to 23 months of age, they identified specific oxygen levels that disrupt the blood-brain barrier (BBB)—a critical structure that protects brain tissue from harmful substances.

This shows a person and a brain.
The BBB is essential for maintaining brain health. In this study, mild and prolonged hypoxia—called chronic mild hypoxia—was found to compromise the BBB in mice. Credit: Neuroscience News

The findings are important for understanding age-related cognitive decline and the potential risks faced by individuals with chronic oxygen-limiting conditions such as asthma, sleep apnea, emphysema, and heart disease.

The BBB is essential for maintaining brain health. In this study, mild and prolonged hypoxia—called chronic mild hypoxia—was found to compromise the BBB in mice. Older mice showed significantly more BBB disruption than younger ones.

Notably, barrier weakening and blood vessel changes occurred at oxygen levels of just 15% in aged mice, compared to 13% in young mice. These data suggest that the aging brain is more sensitive to oxygen deprivation, even at levels that may be considered only mildly hypoxic.

The researchers also determined when this vulnerability emerged. The BBB showed increased sensitivity to low oxygen not only in aged mice but also in mice as young as 2 to 6 months, with a second spike occurring between 12 and 15 months—equivalent to middle age in mice.

These findings may reflect age-dependent changes in brain vascular function and remodeling capacity.

“Hypoxia-induced endothelial proliferation was relatively constant across the age range, but advanced age strongly enhanced the degree of BBB disruption (4-6-fold greater in 23 months vs. 2 months old).”

Another key focus was microglial activation, a sign of brain inflammation. Aged mice exhibited higher microglial activation across all oxygen levels, including normal conditions. Chronic microglial activation is closely linked to neuroinflammation and has been implicated in diseases such as Alzheimer’s.

While the rate of blood vessel formation was constant across ages, the degree of BBB disruption increased sharply with age, suggesting that repair mechanisms may weaken over time.

These results may help explain why older adults with chronic hypoxia-related diseases are at higher risk for neurodegeneration and cognitive decline. The study also draws attention to the risks of high-altitude exposure for aging populations, where oxygen levels naturally drop.

Altogether, these findings underscore the importance of protecting brain health in older individuals by managing oxygen exposure and reducing hypoxia-related risks. The researchers emphasize the need to develop new therapies that support blood-brain barrier integrity, particularly in aging populations exposed to chronic or intermittent low-oxygen conditions.

About this neuroscience and hypoxia research news

Author: Ryan Braithwaite
Source: Impact Journals
Contact: Ryan Braithwaite – Impact Journals
Image: The image is credited to Neuroscience News

Original Research: Open access.
Defining the hypoxic thresholds that trigger blood-brain barrier disruption: the effect of age” by Arjun Sapkota et al. Aging

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