Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Sunday, October 19, 2025

Astrocytes in maintaining neuronal health and brain function: interplay of aging, diet, and environment

 How is your competent? doctor ENSURING YOUR ASTROCYTES ARE WORKING CORRECTLY?  Hasn't even considered that as needed? Fire them!

  • astrocytes (108 posts to June 2011)
  • Astrocytes in maintaining neuronal health and brain function: interplay of aging, diet, and environment

    Abstract

    Astrocytes are pivotal modulators of neuronal health and brain function through their roles in metabolic support, synaptic regulation, neurotransmitter recycling, and the maintenance of the blood–brain barrier. However, aging and environmental challenges compromise astrocytic function, setting the stage for neurodegeneration. Recent findings reveal that age-related astrocyte senescence—characterized by mitochondrial decline, structural atrophy, and a pro-inflammatory shift—undermines their capacity to support neurons, leading to cognitive decline and neurodegenerative conditions such as Alzheimer’s disease. Environmental factors, notably dietary influences, further modulate astrocytic behavior. High-fat diets may initially enhance aspects of astrocytic function, such as glutamate clearance; yet prolonged exposure often triggers maladaptive metabolic shifts and neuroinflammation. In contrast, caloric restriction promotes metabolic flexibility and exerts anti-inflammatory effects, thereby preserving astrocytic integrity. Sleep also plays a crucial role by facilitating glymphatic clearance and synaptic maintenance, whereas sleep deprivation disrupts calcium signaling and exacerbates inflammatory processes. This review synthesizes recent advances in the metabolic, immune, and intercellular mechanisms underlying astrocytic dysfunction in aging. By integrating these insights, we highlight the therapeutic potential of targeting astrocyte-mediated processes to preserve cognitive resilience and counteract neurodegeneration.

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