Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, November 16, 2016

Rotational Atherectomy in Clinical Practice

I would be really concerned about the cutting blades nicking the arterial walls and causing damage, leading to clots being thrown off into the brain. But I have no medical training and thus can't comment on dangers with this. Will your doctor guarantee that the <10 µm particle size will not cause a stroke?
http://circinterventions.ahajournals.org/content/9/11/e004571.extract?etoc
Goran Stankovic and Dejan Milasinovic
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Unlike balloon dilation that results in the displacement of atherosclerotic plaque with multiple intimal tears, rotational atherectomy (RA) is based on the principle of differential cutting that allows for physical removal of inelastic atherosclerotic material while rendering the inner lumen surface smooth.1 Although plaque reduction by pulverization of atherosclerotic material into <10 µm particles has remained its central paradigm,2 the conceptual framework has shifted from the original approach of RA as a debulking strategy and, thus, applicable in a broad array of coronary lesions with large plaque burden, to a contemporary selective clinical utilization, with an emphasis mainly on plaque modification prior to stent implantation.3,4 This transition in conceptual understanding of the targeted effects of RA has been mirrored by a decreasing tendency in its use, from 20% in the mid 1990s to 1% to 3% according to contemporary reports.4,5 Today, RA is used selectively, mainly to disrupt the continuity of the calcium ring within the vessel wall and, thus, facilitate optimal drug-eluting stent (DES) implantation in patients with severely calcified de novo coronary lesions. ROTAXUS (Rotational Atherectomy Prior to Taxus Stent Treatment for Complex Native Coronary Artery Disease), as the only randomized trial to date that tested the strategy of routine lesion preparation with RA followed by DES implantation against stenting without RA, showed a higher rate of procedural success in patients undergoing RA, which, however, did not translate in long-term clinical benefit.6 These findings coincided with previous nonrandomized studies that had also supported RA …
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