Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Sunday, January 29, 2017

The type, not just the amount, of sugar consumption matters in risk of health problems

Is your doctor stuck in the past and still focusing on salt?

Paper Raises More Questions About Salt Restriction In Heart Failure

 

Low-Salt Diet Ineffective, Study Finds. Disagreement Abounds.

 

The wrong white crystals: not salt but sugar as aetiological in hypertension and cardiometabolic disease

The latest here:
The type, not just the amount, of sugar consumption matters in risk of health problems


American Physiological Society News
Long–term fructose consumption impairs vascular and liver function in rats.
The type of sugar you eat – and not just calorie count – may determine your risk for chronic disease. A new study is the first of its kind to compare the effects of two types of sugar on metabolic and vascular function. The paper was published ahead of print in the American Journal of Physiology–Heart and Circulatory Physiology.

Female rats were given a liquid solution of either glucose or fructose in addition to their normal diet of solid food. The rats received the sweetened solutions for eight weeks, roughly equivalent to a person eating large amounts of sugar for six years. The sugar–fed rats were compared with a control group that received plain drinking water in addition to their food supply.

Researchers found that although both sugar–fed groups consumed more calories than the control group, the total calorie intake of the glucose–fed rats was higher than the rats that were given fructose. Another surprising observation was that “despite this difference, only the fructose group exhibited a significant increase in final body weight,” wrote the research team.

In addition to higher weight gain, the fructose group showed more markers of vascular disease and liver damage than the glucose group. These included high triglycerides, increased liver weight, decreased fat burning in the liver and impaired relaxation of the aorta, which can affect blood pressure.

These findings suggest that an increase in the amount of calories consumed due to sweeteners is not the only factor involved in long–term health risks. The type of sugar may also play a role in increasing risk factors for heart disease, diabetes and other chronic diseases.

The article is titled “Type of Supplemented Simple Sugar, Not Merely Calorie Intake, Determines Adverse Effects on Metabolism and Aortic Function in Female Rats.”

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