Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Showing posts with label trigeminal nerve stimulation. Show all posts
Showing posts with label trigeminal nerve stimulation. Show all posts

Wednesday, July 15, 2026

‘Neural tourniquet’ device could hold key to stopping life-threatening hemorrhage

 Ask your competent? doctor if this would include brain hemorrhages and the ones from Hemorrhagic transformation ! 

Your doctor is already familiar with this use, right?

NO? So you have a fuckingly incompetent doctor then!

‘Neural tourniquet’ device could hold key to stopping life-threatening hemorrhage

An ear-based wearable device that taps the body’s longest cranial nerve can boost blood clotting in what researchers are hopeful could be a new way of treating severe bleeding. 

That’s according to new data out from Northwell Health’s Feinstein Institutes for Medical Research and published in Bioelectronic Medicine this month. 

Scientists from the Institute unveiled data from the first-in-human mechanistic study suggesting that electrical stimulation to the ear could help healthy adults improve their blood clotting. 

The central premise is that activating the vagus nerve can work as a “neural tourniquet,” as this nerve influences many bodily functions, including inflammation and blood clotting.

The tech comes from Spark Biomedical’s transcutaneous auricular neurostimulation (tAN) platform, which is set up to engage the vagus and trigeminal nerves through the ear.

This first study assessed two auricular stimulation modalities: transcutaneous auricular vagus nerve stimulation (taVNS) and tAN, which combines vagal and trigeminal nerve stimulation. 

The researchers found that both approaches successfully activated platelets, increasing their readiness to respond to injury. 

“Specifically, taVNS accelerated the initiation, propagation, and stabilization of blood clots,” the Institute said in a release.

The data are early stage, and further trials will be needed to validate the technology's worth.

The scientists behind the study are hailing the approach as a potentially new, non-invasive method of helping in severe bleeding situations, such as from blunt trauma, on the battlefield, or in hemophilia patients.  

The device is small and can be carried as part of a medical kit, making it easy to use. 

“Imagine a non-painful technology that taps into the body’s own healing powers and saves a person from bleeding to death,” said Jared Huston, M.D., professor of Surgery and Science Education in the Department of Surgery at Northwell and the Institute of Bioelectronic Medicine at the Feinstein Institutes, in a statement. 

“With this study, we have more confidence that bioelectronic medicine is safe to use for the treatment of bleeding conditions and I hope to conduct additional studies to develop this potential treatment," he said. 

Thursday, August 3, 2017

Researchers find neuroprotective effects of trigeminal nerve stimulation in severe TBI

Would this help in stroke? Better oxygenation and CBF sounds useful. We'll never know.
https://www.news-medical.net/news/20170728/Researchers-find-neuroprotective-effects-of-trigeminal-nerve-stimulation-in-severe-TBI.aspx
Researchers at the Feinstein Institute for Medical Research and the department of neurosurgery at the Hofstra Northwell School of Medicine, announced today that they have published a paper with research findings that could have implications for the treatment of many neurological conditions, including severe traumatic brain injury (TBI). The team of researchers found that in an animal model with TBI, trigeminal nerve stimulation (TNS) resulted in increased cerebral blood flow (CBF) and oxygen to the brain. These latest findings were published in Scientific Reports.
The research paper titled, "Neuroprotective Effects of Trigeminal Nerve Stimulation in Severe Traumatic Brain Injury," was co-authored by Northwell Health's Amrit Chiluwal, MD, Raj K. Narayan, MD, Wayne Chaung, PhD, Neal Mehan, MD, Ping Wang, MD, Chad E. Bouton, and Chunyan Li, PhD. The paper was also co-authored by Eugene V. Golanov, MD, PhD, from the department of neurosurgery at the Houston Methodist Research Institute in Houston, TX.
"Following TBI, ischemia and hypoxia play a major role in further worsening of the damage, a process known as secondary injury," said Dr. Li, assistant professor of the Center for Bioelectronic Medicine at the Feinstein Institute. "Preventing secondary injury is vitally important in the overall management of TBI. In the animal model, we investigated the use of electrical TNS for improving CBF and delivering more oxygen to the brain, with the goal of decreasing secondary injury. We found that TBI rat models with TNS treatment demonstrated significantly increased systemic blood pressure, CBF, oxygen, as well as significantly reduced brain edema, blood-brain barrier disruption and lesion volume."
Dr. Narayan, Northwell Health's senior vice president and executive director, neurosurgery services added, "No pharmacological agents have currently been shown to improve clinical outcomes for TBI. Therefore, there is an urgent need for developing novel therapeutic strategies to maximize recovery. The data from this research study provides strong evidence that TNS offers neuroprotection following brain damage. TNS could also offer some benefit in other pathological states such as stroke or vasospasm after subarachnoid hemorrhage where the brain is at risk for ischemic and/or inflammatory damage."