Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Friday, June 3, 2022

Walking Speed Helps Predict Future Dementia

 

Your doctor is responsible to get you recovered enough to do a high enough walking speed. YOUR DOCTOR'S RESPONSIBILITY.  Don't let them weasel out of responsibility by quoting this shitworthy statement: 'All strokes are different, all stroke recoveries are different'. Laugh maniacally in their face if they use such crapola.

 

Walking Speed Helps Predict Future Dementia

A motion blurred photo of a senior man walking and a young boy running on a city sidewalk.

A combined drop in both walking speed and cognitive function was tied to an increased risk of dementia, a study of nearly 17,000 older adults showed.

Dual decline in gait speed and cognition carried a higher risk of dementia than either gait-only decline or cognitive-only decline, reported Taya Collyer, PhD, of Monash University in Victoria, Australia, and co-authors, in JAMA Network Open.

Dementia risk was highest in people who had slower gait speeds coupled with lower memory scores over time.

"Slowing gait and failing memory may be the best combination of clinical measures to identify people at risk of future dementia," co-author Michele Callisaya, PhD, of the National Center for Healthy Aging at Monash University and Peninsula Health, told MedPage Today.

"By the time a diagnosis of dementia is made, there's already been a substantial buildup of pathology in the brain," Callisaya said. "It's important to identify at-risk individuals early to address modifiable risk factors for dementia prevention and start new treatments as they become available."

"Gait speed is quick to measure and only requires a measured distance and a stopwatch," she pointed out. "Slowing of more than 0.05 meters per second per year should trigger a more comprehensive assessment."

Gait evaluations often are not performed in clinics, noted Joe Verghese, MD, of Albert Einstein College of Medicine in New York City, in an invited commentary.

"Despite the established predictive validity of gait assessments for geriatric syndromes, an implementation barrier for routine gait assessment in clinics exists that needs to be addressed to improve care of older patients," Verghese observed.

Traditionally, gait dysfunction has not been considered an early clinical feature of Alzheimer's disease and was seen more as a marker of non-Alzheimer's pathology, Verghese noted.

"However, systematic longitudinal studies and finer-grain analysis of gait performance in the context of cognitive decline is calling this view into question," he wrote. A recent exploratory study suggested Alzheimer's and Lewy body disease have unique signatures of gait impairment that may reflect underlying pathology, for example.

Collyer and colleagues analyzed data from 16,885 participants in the ASPREE trial of low-dose aspirin, which ran from 2010 to 2017 in the U.S. and Australia. Mean age of participants was 75 and 56% were women.

At baseline and at years 2, 4, 6, and study close-out, participants had gait speed measured by completing two walks of 3 meters each. Gait decline was defined as a drop in speed of at least 0.05 meters/second/year across the study.

At baseline and years 1, 3, 5, and close-out, participants underwent four different cognitive assessments: the Modified Mini-Mental State Examination to measure global cognitive function, the Hopkins Verbal Learning Test Revised to assess memory, the Controlled Oral Word Association Test for verbal fluency, and the Symbol Digit Modalities Test for processing speed. Cognitive decline was defined as the the lowest tertile of annual change.

Compared with non-decliners, dementia incidence was highest in people who had declines in both gait and memory (HR 24.7), followed by gait and global cognition (HR 22.2), gait and verbal fluency (HR 4.7), and gait and processing speed (HR 4.3).

"Importantly, we answer the question as to whether dual decline in gait speed and a test of processing speed or verbal fluency exhibits a similar association with progression to dementia as decline in memory (delayed recall) or global cognition," Collyer and co-authors wrote.

The analysis had a few limitations, the researchers noted. Gait and cognition were measured at separate time points throughout the study. ASPREE participants also were healthier than many older adults, and results might not apply to other populations.

  • Judy George covers neurology and neuroscience news for MedPage Today, writing about brain aging, Alzheimer’s, dementia, MS, rare diseases, epilepsy, autism, headache, stroke, Parkinson’s, ALS, concussion, CTE, sleep, pain, and more. Follow

Disclosures

The ASPREE trial was funded by the National Institute on Aging in the U.S. and National Health and Medical Research Council in Australia.

Researchers reported relationships with NIH, Victorian Cancer Agency, Biogen, Roche, National Health and Medical Research Council, Neurology, International Journal of Stroke, Amylyx Pharmaceuticals, Athira Pharma, Eli Lilly, Genentech, Novartis, and the Alzheimer's Association.

The editorialist reported relationships with NIH, Catch-U, and MedRhythms.

 

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