Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Tuesday, December 20, 2022

Dementia Risk Higher in People With Earlier Signs of Psychological Distress

Post stroke, already a major stress event, then getting fired, moving a couple of states away, getting divorced. All which should have left me a blubbering pile of Jello. Yet, the stroke was the best thing to happen to me, it got me divorced. I'm happy now. I won't be having another stroke or dementia.

Why my stroke was the best thing to ever happen to me

The latest here:

Dementia Risk Higher in People With Earlier Signs of Psychological Distress

Stress, exhaustion symptoms linked with increased dementia risk years later

A portrait of a stressed looking mature man with his hands on his head

Dementia risk was higher in people who had signs of psychological distress earlier in life, a cohort study in Finland found.

In more than 67,000 people with an average age of 45, self-reported distress symptoms -- notably, stress, and exhaustion -- were associated with a 17% to 24% increased risk of dementia over a 25-year follow-up period, reported Sonja Sulkava MD, PhD, of the Finnish Institute for Health and Welfare in Helsinki, and co-authors.

These self-reported symptoms were tied to an 8% to 12% increase in lifetime dementia after accounting for the competing risk of death, which was more common than dementia over time, Sulkava and colleagues wrote in JAMA Network Openopens in a new tab or window.

"Our study suggests symptoms of psychological distress like exhaustion, depressive mood, and the experience of stress are risk factors for dementia, and not only prodromal symptoms of underlying dementia disorder," Sulkava wrote in an email to MedPage Today.

"Previous studies have shown an association between symptoms of psychological distress and dementia, but the nature of the association is still unclear," Sulkava wrote. "We clarified that connection using a large population data set with 10 to 45 years of follow-up and careful modeling of death for other causes."

The study used data from 67,688 people who completed National FINRISK Study surveys between 1972 and 2007. Participants self-reported symptoms of psychological distress in the prior month, including whether they experienced stress more than other people and whether they experienced depressive mood, exhaustion, and nervousness often, sometimes, or never. The cohort was linked to the Finnish Health Register for dementia and mortality data.

About half (51.7%) of participants were women. Baseline ages ranged from 25 to 74 years, with a mean age of 45.4. Findings were adjusted for age, sex, baseline year, follow-up time, educational level, BMI, smoking, diabetes, systolic blood pressure, cholesterol, and physical activity.

Over a mean follow-up of 25.4 years, 7,935 participants received a diagnosis of dementia. The competing risk of death was more common, occurring in 19,647 people.

In a Poisson cause-specific model, exhaustion was linked with subsequent all-cause dementia (incidence rate ratio [IRR] 1.17, 95% CI 1.08-1.26), as was stress (IRR 1.24, 95% CI 1.11-1.38). These relationships remained significant in sensitivity analyses.

A subdistribution hazard model that estimated the effect on cumulative incidence also showed exhaustion (HR 1.08, 95% CI 1.01-1.17) and stress (HR 1.12, 95% CI 1.00-1.25) were linked with dementia risk over time. In contrast, depressive mood (HR 1.08, 95% CI 0.98-1.20) did not show a statistically significant association.

The study of psychological distress and brain health is "far from being incisive," noted Yoram Barak, MD, MHA, psychiatrist of the University of Otago in Dunedin, New Zealand, in an accompanying editorialopens in a new tab or window.

This "sophisticated analysis" adds "an important facet to the field by accounting for competing risk of death," Barak observed. "This should become a standard when researching these questions."

But relying on survey questions about experiences in the previous month may negate the possibility of understanding how stress may be a causative agent in dementia, since no information about lifelong traits or clinically diagnosed anxiety or depression was collected, Barak pointed out.

"We need to advance the field farther by creatively studying lifelong patterns of emotional states and relationships," he wrote. "Life trajectories of individuals and couples will teach us more about stress, distress, tensity, neuroticism, and dementia."

While the study questions on psychological distress do not form a validated multi-item questionnaire, the one-item measures for different symptoms of psychological distress correlate significantly, Sulkava and colleagues noted.

The research had other limitations, they acknowledged. Data about traumatic brain injury, hearing impairment, and low social contact -- three known dementia risk factors -- were not available. In addition, participants with missing covariate information had more risk factors for dementia or mortality.

  • Judy George covers neurology and neuroscience news for MedPage Today, writing about brain aging, Alzheimer’s, dementia, MS, rare diseases, epilepsy, autism, headache, stroke, Parkinson’s, ALS, concussion, CTE, sleep, pain, and more. Follow

Disclosures

This study was supported by the Emil Aaltonen Foundation, Maud Kuistila Memorial Foundation, Academy of Finland, and Gyllenberg Foundation.

Sulkava reported grants from Emil Aaltonen Foundation, Maud Kuistila Memorial Foundation, Gyllenberg Foundation; and a family member employed by Amialife Ltd.

Barak reported no disclosures.

Primary Source

JAMA Network Open

Source Reference: opens in a new tab or windowSulkava S, et al "Association between psychological distress and incident dementia in a population-based cohort in Finland" JAMA Netw Open 2022; DOI: 10.1001/jamanetworkopen.2022.47115.

Secondary Source

JAMA Network Open

Source Reference: opens in a new tab or windowBarak Y "Stress, distress, tensity, neuroticism, and risk of dementia" JAMA Netw Open 2022; DOI: 10.1001/jamanetworkopen.2022.47124.

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