Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Monday, July 13, 2026

Biomechanical and neural correlates of FastFES versus Fast gait training in individuals post stroke: a randomized control trial study protocol

 You're going to have to cure a lot of survivors of spasticity before you can even get to fast gait training! If I walk fast my left knee hyperextends and snaps, eventually knee replacement will be needed, all because of not doing anything about my spasticity.

30% get spasticity

Biomechanical and neural correlates of FastFES versus Fast gait training in individuals post stroke: a randomized control trial study protocol


  • 1. Division of Physical Therapy, Department of Rehabilitation Medicine, Emory University School of Medicine, Atlanta, GA, United States

  • 2. Department of Physical Therapy, College of Allied Health Sciences, University of Illinois Chicago, Chicago, IL, United States

Abstract

Background: 

Fast gait training, individually and when combined with functional electrical stimulation (FastFES), has been shown to improve walking function in individuals post stroke. However, the neural mechanisms underlying the effects of these two gait training interventions are poorly understood. The purpose of this mechanism-focused gait rehabilitation randomized clinical trial is to assess the effects of Fast and FastFES gait training interventions on corticospinal neurophysiology, gait biomechanics, energy cost, and walking function in individuals with chronic post-stroke hemiparesis.

Methods: 

In this randomized clinical trial, participants with chronic stroke are recruited and randomized to receive one of two gait training interventions—FastFES or Fast. Participants in each intervention group receive 12 sessions of gait training, with each training session comprising 30 min of training. During FastFES training, electrical stimulation is delivered to ankle dorsi- and plantar-flexor muscles during paretic swing phase and late stance phase, respectively. Evaluations of clinical, gait biomechanics, neurophysiological, and energy cost outcomes are performed at baseline, after completion of 12 training session (post12), and at 3-weeks and 6-weeks after completion of training (3-week follow up, 6-week follow up), to measure longitudinal effects of gait training. Additional evaluations are performed at completion of 3 and 6 training sessions (post3 and post6) to measure the time course of change during gait training. Upon completion of the study, planned analyses will include between-group comparisons of FastFES versus Fast gait training on training-induced changes in corticomotor and spinal excitability, gait biomechanics outcomes such as peak anterior ground reaction force, as well as association of training-induced changes in corticospinal neurophysiology and gait biomechanics with clinical and energy cost measures.

Discussion: 

By elucidating the biomechanical and neural correlates underlying gait training-induced changes in locomotor function, this study promises to build on existing evidence supporting the clinical effects of FastFES and Fast gait training. The long-term goal of this study is to inform the development of neurobiology-informed, personalized, and innovative strategies to enhance the effectiveness of stroke gait rehabilitation.

Clinical trial registration:

clinicaltrials.gov, identifier NCT04380454.

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