Part of the reason for my extreme fatigue post stroke was sleep apnea which my hospital doctors never found. Once found, the CPAP machine was useless, couldn't sleep with the need to forcibly exhale against the incoming air. Have no clue if I still have sleep apnea, no partner to tell me otherwise, my fatigue has lessened considerably so I'm guessing my sleeping habits are now preventing the problem.
Does Obstructive Sleep Apnea Increase Dementia Risk?
Obstructive sleep apnea (OSA) may be associated with poorer memory performance and higher estimated risk for future dementia, according to study findings published in Alzheimer’s & Dementia.
Researchers evaluated whether self-reported OSA was associated with cognitive function and dementia risk among cognitively unimpaired adults in midlife and whether apolipoprotein E (APOE) ε4 carrier status modified those associations.
The researchers conducted a cross-sectional analysis of baseline data from the Healthy Brain Project, an Australian community-based cohort enriched for individuals with a family history of dementia. The analysis included 2795 cognitively unimpaired adults aged 40 to 70 years. OSA status was determined by self-report, cognitive performance was assessed with the Cogstate Brief Battery, and dementia risk was estimated using the Cardiovascular Risk Factors, Aging and Incidence of Dementia (CAIDE) score.
These findings highlight the need for early OSA screening to identify individuals at elevated dementia risk. Participants had a mean age of 56.4 years, 76% were women, and 7% reported an OSA diagnosis. Approximately one-third carried the APOE ε4 allele, more than one-half reported a first- or second-degree family history of dementia, and nearly three-fifths of those with OSA were receiving treatment. Compared with participants without OSA, those with OSA were older, had higher body mass index values, and were more likely to be men and have hypertension or hypercholesterolemia. They also reported poorer sleep quality, greater daytime sleepiness, more severe insomnia symptoms, and higher anxiety and depression scores. After adjustment for demographic characteristics and OSA treatment status, participants with OSA demonstrated significantly poorer memory performance than those without OSA (Cohen d, -0.22; P =.03), although attention did not differ between groups.Participants with OSA also had significantly higher CAIDE risk scores than those without OSA (Cohen d, 0.84; P <.001). Elevated risk persisted when analyses were limited to the modifiable vascular components of the CAIDE score (Cohen d, 0.72; P <.001), including obesity, hypertension, hypercholesterolemia, and physical inactivity.
APOE ε4 carrier status did not significantly modify the associations between OSA and either cognitive performance or dementia risk. Participants with OSA had higher CAIDE risk scores regardless of APOE ε4 status, whereas APOE ε4 carriage alone was not associated with higher CAIDE scores.
Additional analyses suggested that poorer memory performance was confined to participants with untreated OSA. Memory performance among treated participants did not differ significantly from that of participants without OSA, and the association between OSA and memory was no longer significant after adjustment for vascular risk burden.
Study limitations include reliance on self-reported OSA diagnoses, limited information on treatment type and adherence, use of a brief cognitive assessment rather than a comprehensive neuropsychological battery, and the cross-sectional design, which precluded assessment of temporal relationships or causality.
“These findings highlight the need for early OSA screening to identify individuals at elevated dementia risk,” the study authors concluded.
Disclosures: This research was supported by the National Health and Medical Research Council, the Alzheimer’s Association, the Dementia Australia Research Foundation, the Bethlehem Griffiths Research Foundation, the Yulgilbar Alzheimer’s Research Program, the National Heart Foundation of Australia, and the Australian Government Research Training Program Scholarship. Please see the original reference for a full list of disclosures.
References:
Abdelmessih GT, Bransby L, Cummins H, Jackson ML, Lim YY. Associations of self-reported obstructive sleep apnea with cognition and dementia risk in cognitively unimpaired middle-aged adults. Alzheimers Dement. 2026;22(7):e71553. doi:10.1002/alz.71553
No comments:
Post a Comment