Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Saturday, August 1, 2026

The aging brain that doesn’t fail: how neural resilience masks neurodegeneration

Have your competent? doctor provide you EXACT PROTOCOLS for cognitive reserve and resilience. A competent? doctor would have distributed the recovery protocol that Pedro Bach-y-Rita  used to recover fully back in 1958 with only a partial brain! But you don't have a competent doctor, do you? Hasn't done anything so you can be like Bernadette the nun? Lost the link but your doctor should know about her.

NO? So, willing to DO NOTHING, LIKE USUAL!

cognitive reserve (51 posts to November 2014)
cognitive resilience (18 posts to September 2014)

cognitive rehabilitation (34 posts to December 2015)

 The aging brain that doesn’t fail: how neuralresilience masks neurodegeneration

Andrew Eisen , Heather D. Durham & Erik Pioro To cite this article: Andrew Eisen , Heather D. Durham & Erik Pioro (27 Jul 2026): The aging brain that doesn’t fail: how neural resilience masks neurodegeneration, Amyotrophic Lateral Sclerosis and Frontotemporal Degeneration, DOI: 10.1080/21678421.2026.2705693 To link to this article: https://doi.org/10.1080/21678421.2026.2705693
ANDREW EISEN 1 2 1 , HEATHER D. DURHAM 2 & ERIK PIORO 1 Division of Neurology, Department of Medicine, University of British Columbia, Vancouver, British Columbia, Canada, and Department of Neurology and Neurosurgery, Montreal Neurological Institute, McGill University, Montreal, Quebec, Canada 

Abstract 


 Neurodegenerative diseases such as amyotrophic lateral sclerosis, Alzheimer’s disease, and Parkinson’s disease are usually framed as consequences of aging-related pathogenic processes, including impaired proteostasis with protein aggregation, mitochondrial dysfunction, oxidative stress, and neuroinflammation. Yet, most individuals, even into advanced age, do not develop clinically significant neurodegenerative disease. This discrepancy suggests that the nervous system possesses robust and redundant protective mechanisms that maintain neural integrity despite cumulative molecular and cellular stress. In this perspective, we propose that neurodegenerative diseases arise not simply from the presence of pathogenic processes, but when integrated resilience systems fail to maintain homeostasis or when reserve mechanisms no longer compensate for accumulated pathology. We have synthesized a threshold-based model of disease emergence based on evidence across proteostasis, mitochondrial function, neuroimmune regulation, glial biology, network-level compensation, and barrier integrity, while integrating genetic, environmental, developmental, and stochastic modifiers. We distinguish biological resilience, which actively limits or repairs pathology, from reserve, which permits function despite pathology. We further propose that clinical disease emerges only when age-related cumulative stress exceeds the combined capacity of resilience and reserve. Reframing neurodegeneration as a failure of preservation systems offers new directions for pre vention and therapeutic development. 

Keywords: Aging, neurodegeneration, senescence, resilience 

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