Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, March 11, 2026

Marijuana compound may help prevent dementia when paired with common drug

 Will your competent? doctor put 2 and 2 together and see if this could prevent your increased risk of dementia post stroke? Or is your doctor so incompetent that knowledge of this doesn't even exist?

Didn't this from November 2013 already lead the way?

Your risk of dementia, has your doctor told you of this?  Your doctor is responsible for preventing this! Is s/he willing to try this on you?

1. A documented 33% dementia chance post-stroke from an Australian study?   May 2012.

2. Then this study came out and seems to have a range from 17-66%. December 2013.`    

3. A 20% chance in this research.   July 2013.

4. Dementia Risk Doubled in Patients Following Stroke September 2018 

The latest here:

Marijuana compound may help prevent dementia when paired with common drug

THC shown to keep Alzheimer's at bay when combined with pain medication

As marijuana's potential health benefits are under debate, new research has found that THC could have a powerful and positive effect on the brain.

Researchers at the University of Texas (UT) at San Antonio, Long School of Medicine, recently discovered that THC (tetrahydrocannabinol, the main psychoactive compound in marijuana) could prevent the development of Alzheimer’s disease – but only when paired with an anti-inflammatory drug called celecoxib.

Celecoxib is a selective COX-2 inhibitor that is commonly prescribed for arthritis and pain.

ALZHEIMER’S DECLINE COULD SLOW DRAMATICALLY WITH ONE SIMPLE DAILY HABIT, STUDY FINDS

While THC has previously been shown to have anti-inflammatory and neuroprotective properties, it’s also been linked to negative effects on the brain, particularly in learning and memory, according to a UT Health press release.

Combining THC with an anti-inflammatory drug, however, could provide the same benefits with fewer negative side effects.

THC cannabis drops

THC could be powerful against cognitive decline when combined with an anti-inflammatory, a new study suggests. (iStock)

The study, which was published in the journal Aging and Disease, paired low-dose THC extract with celecoxib, which was administered to mice daily for 30 days.

The mice received the treatments before the development of memory symptoms to measure the impact on preventing or delaying Alzheimer’s.

The results showed improved cognition, learning and memory, as well as decreased markers of neuroinflammation and reduced Alzheimer’s-related brain pathology, the release stated.

Although THC alone had the same results, it also increased inflammatory signals, while the combination dosage did not.

"What really mattered was behavior. If cognition is not improved, then the treatment doesn’t matter. And that’s where the combination clearly worked better than THC alone," noted lead study author Chu Chen, Ph.D., professor in the Department of Cellular and Integrative Physiology.

Years of research led Chen to connect the dots on how THC can manipulate the brain and why it’s difficult to use safely for neurological conditions, according to UT Health.

"When THC is given, it unexpectedly increases COX-2 in the brain. That increase is closely associated with learning and memory impairment," Chen said.

Both THC and celecoxib are FDA-approved for use in humans, which could provide a "real advantage" in moving along to clinical trials, according to UT Health.

"What really mattered was behavior. If cognition is not improved, then the treatment doesn’t matter."

Future studies will investigate whether the drug combination can slow disease progression or reverse deficits after symptoms have appeared.


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